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However, the mechanism involved in <jats:styled-content style=\"fixed-case\">IMAT</jats:styled-content> formation from these progenitor cells <jats:italic>in vivo</jats:italic> remains unclear. In the present study, among the growth factors tested, which were known to be expressed in skeletal muscle, we found only basic fibroblast growth factor (<jats:styled-content style=\"fixed-case\">bFGF</jats:styled-content>) has a pro‐adipogenic effect on skeletal muscle derived adipogenic progenitor clone, <jats:styled-content style=\"fixed-case\">2G11</jats:styled-content> cells. Pre‐exposure of <jats:styled-content style=\"fixed-case\">2G11</jats:styled-content> cells to <jats:styled-content style=\"fixed-case\">bFGF</jats:styled-content> did not affect initial gene expressions of CCAAT/enhancer‐binding protein (<jats:styled-content style=\"fixed-case\">C</jats:styled-content>/<jats:styled-content style=\"fixed-case\">EBP)</jats:styled-content>β and <jats:styled-content style=\"fixed-case\">C</jats:styled-content>/<jats:styled-content style=\"fixed-case\">EBP</jats:styled-content>δ, while resulting in an enhancement of subsequent expressions of <jats:styled-content style=\"fixed-case\">C</jats:styled-content>/<jats:styled-content style=\"fixed-case\">EBP</jats:styled-content>α and proliferator‐activated receptor gamma (<jats:styled-content style=\"fixed-case\">PPAR</jats:styled-content>γ) during adipogenesis, indicating that <jats:styled-content style=\"fixed-case\">bFGF</jats:styled-content> is acting on the transcriptional regulation of <jats:styled-content style=\"fixed-case\">C</jats:styled-content>/<jats:styled-content style=\"fixed-case\">EBP</jats:styled-content>α and <jats:styled-content style=\"fixed-case\">PPAR</jats:styled-content>γ. In addition, the effect of <jats:styled-content style=\"fixed-case\">bFGF</jats:styled-content> is mediated via two types of <jats:styled-content style=\"fixed-case\">FGF</jats:styled-content> receptor (<jats:styled-content style=\"fixed-case\">FGFR</jats:styled-content>) isoforms: <jats:styled-content style=\"fixed-case\">FGFR1</jats:styled-content> and <jats:styled-content style=\"fixed-case\">FGFR2 IIIc</jats:styled-content>, and both receptors are prerequisite for <jats:styled-content style=\"fixed-case\">bFGF</jats:styled-content> to express its pro‐adipogenic effect. These results suggest that <jats:styled-content style=\"fixed-case\">bFGF</jats:styled-content> plays an important role as a key trigger of <jats:styled-content style=\"fixed-case\">IMAT</jats:styled-content> formation <jats:italic>in vivo</jats:italic>.</jats:p>"}]}],"creator":[{"@id":"https://cir.nii.ac.jp/crid/1380861286772638209","@type":"Researcher","foaf:name":[{"@value":"Shin‐ichi Nakano"}],"jpcoar:affiliationName":[{"@value":"Department of Veterinary Physiology Graduate School of Agricultural and Life Sciences The University of Tokyo  Tokyo Japan"}]},{"@id":"https://cir.nii.ac.jp/crid/1380004235478286863","@type":"Researcher","foaf:name":[{"@value":"Katsuyuki Nakamura"}],"jpcoar:affiliationName":[{"@value":"Department of Veterinary Physiology Graduate School of Agricultural and Life Sciences The University of Tokyo  Tokyo Japan"}]},{"@id":"https://cir.nii.ac.jp/crid/1380004235478286862","@type":"Researcher","foaf:name":[{"@value":"Naomi Teramoto"}],"jpcoar:affiliationName":[{"@value":"Department of Veterinary Physiology Graduate School of Agricultural and Life Sciences The University of Tokyo  Tokyo 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