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Are Dysregulated Inflammatory Responses to Commensal Bacteria Involved in the Pathogenesis of Hepatobiliary-Pancreatic Autoimmune Disease? An Analysis Using Mice Models of Primary Biliary Cirrhosis and Autoimmune Pancreatitis
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- Naoko Yanagisawa
- Departments of Microbiology and Immunology, Tokyo Women’s Medical University, 8-1 Kawada-cho, Shinjuku-ku, Tokyo, Japan
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- Ikuko Haruta
- Departments of Microbiology and Immunology, Tokyo Women’s Medical University, 8-1 Kawada-cho, Shinjuku-ku, Tokyo, Japan
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- Ken Kikuchi
- Department of Infection Control Science, Faculty of Medicine, Juntendo University, Tokyo, Japan
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- Noriyuki Shibata
- Department of Pathology, Tokyo Women’s Medical University, Tokyo, Japan
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- Junji Yagi
- Departments of Microbiology and Immunology, Tokyo Women’s Medical University, 8-1 Kawada-cho, Shinjuku-ku, Tokyo, Japan
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Description
<jats:p>The etiopathogenesis of many autoimmune disorders has not been identified. The aim of this paper is to focus on the involvement of bacterial exposure in the pathogenesis of primary biliary cirrhosis (PBC) and autoimmune pancreatitis (AIP), both of which are broadly categorized as autoimmune disorders involving hepatobiliary-pancreatic lesions. Avirulent and/or commensal bacteria, which may have important role(s) as initiating factors in the pathogenesis of autoimmune disorders such as PBC and AIP, will be emphasized.</jats:p>
Journal
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- ISRN Gastroenterology
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ISRN Gastroenterology 2011 1-8, 2011-06-15
Wiley
