{"@context":{"@vocab":"https://cir.nii.ac.jp/schema/1.0/","rdfs":"http://www.w3.org/2000/01/rdf-schema#","dc":"http://purl.org/dc/elements/1.1/","dcterms":"http://purl.org/dc/terms/","foaf":"http://xmlns.com/foaf/0.1/","prism":"http://prismstandard.org/namespaces/basic/2.0/","cinii":"http://ci.nii.ac.jp/ns/1.0/","datacite":"https://schema.datacite.org/meta/kernel-4/","ndl":"http://ndl.go.jp/dcndl/terms/","jpcoar":"https://github.com/JPCOAR/schema/blob/master/2.0/"},"@id":"https://cir.nii.ac.jp/crid/1360565166012359296.json","@type":"Article","productIdentifier":[{"identifier":{"@type":"DOI","@value":"10.1016/j.kint.2017.10.019"}},{"identifier":{"@type":"URI","@value":"https://api.elsevier.com/content/article/PII:S0085253817307998?httpAccept=text/xml"}},{"identifier":{"@type":"URI","@value":"https://api.elsevier.com/content/article/PII:S0085253817307998?httpAccept=text/plain"}},{"identifier":{"@type":"PMID","@value":"29475546"}}],"resourceType":"学術雑誌論文(journal article)","dc:title":[{"@value":"IgA nephropathy and IgA vasculitis with nephritis have a shared feature involving galactose-deficient IgA1-oriented pathogenesis"}],"description":[{"notation":[{"@value":"Galactose-deficient IgA1 has been proposed as an important effector molecule in IgA nephropathy (IgAN). We previously showed that the galactose-deficient IgA1-specific monoclonal antibody KM55 can detect circulating galactose-deficient IgA1 in patients with IgAN, enabling us to study the molecular roles of galactose-deficient IgA1. Herein, we further examined the pathophysiological significance of galactose-deficient IgA1 in glomerular deposits of patients with IgAN by immunohistochemistry using KM55. Immunostaining of galactose-deficient IgA1 with KM55 was performed in paraffin-embedded sections of renal biopsy specimens from 48 patients with IgAN and 49 patients with other renal diseases such as lupus nephritis, HCV-related nephropathy, IgA vasculitis with nephritis (IgA-VN), and membranous nephropathy. Glomerular galactose-deficient IgA1 was specifically detected in IgAN and IgA-VN but not in the other renal diseases. Galactose-deficient IgA1 was localized predominantly in the mesangial region as IgA deposition. However, galactose-deficient IgA1 was not detected in patients with lupus nephritis accompanied by glomerular IgA deposition. Thus, our study strongly suggests that IgAN and IgA-VN have a shared feature regarding galactose-deficient IgA1-oriented pathogenesis."}]}],"creator":[{"@id":"https://cir.nii.ac.jp/crid/1420845751158647808","@type":"Researcher","personIdentifier":[{"@type":"KAKEN_RESEARCHERS","@value":"10468572"},{"@type":"NRID","@value":"1000010468572"},{"@type":"NRID","@value":"9000005990282"},{"@type":"NRID","@value":"9000398624487"},{"@type":"NRID","@value":"9000415088579"},{"@type":"NRID","@value":"9000402763942"},{"@type":"NRID","@value":"9000309181770"},{"@type":"RESEARCHMAP","@value":"https://researchmap.jp/hyy"}],"foaf:name":[{"@value":"Hitoshi Suzuki"}]},{"@id":"https://cir.nii.ac.jp/crid/1380003444233012736","@type":"Researcher","foaf:name":[{"@value":"Junichi Yasutake"}]},{"@id":"https://cir.nii.ac.jp/crid/1410282679656912131","@type":"Researcher","personIdentifier":[{"@type":"NRID","@value":"9000019195114"},{"@type":"RESEARCHMAP","@value":"https://researchmap.jp/juntendojinzo"}],"foaf:name":[{"@value":"Yuko Makita"}]},{"@id":"https://cir.nii.ac.jp/crid/1380003444233012741","@type":"Researcher","foaf:name":[{"@value":"Yuki Tanbo"}]},{"@id":"https://cir.nii.ac.jp/crid/1380003444233012865","@type":"Researcher","foaf:name":[{"@value":"Kohei Yamasaki"}]},{"@id":"https://cir.nii.ac.jp/crid/1380003444233012739","@type":"Researcher","foaf:name":[{"@value":"Tadashi Sofue"}]},{"@id":"https://cir.nii.ac.jp/crid/1380003444233012738","@type":"Researcher","foaf:name":[{"@value":"Toshiki Kano"}]},{"@id":"https://cir.nii.ac.jp/crid/1420282801189441152","@type":"Researcher","personIdentifier":[{"@type":"KAKEN_RESEARCHERS","@value":"70372935"},{"@type":"NRID","@value":"1000070372935"},{"@type":"NRID","@value":"9000019083452"},{"@type":"NRID","@value":"9000273016503"},{"@type":"NRID","@value":"9000256090958"},{"@type":"NRID","@value":"9000380978493"},{"@type":"NRID","@value":"9000000301149"},{"@type":"NRID","@value":"9000381455620"},{"@type":"NRID","@value":"9000020513812"},{"@type":"NRID","@value":"9000414346507"},{"@type":"NRID","@value":"9000361700398"},{"@type":"NRID","@value":"9000415088577"},{"@type":"NRID","@value":"9000343333470"},{"@type":"NRID","@value":"9000255722233"},{"@type":"NRID","@value":"9000378652447"},{"@type":"NRID","@value":"9000346931995"},{"@type":"NRID","@value":"9000356658855"},{"@type":"NRID","@value":"9000335558611"},{"@type":"RESEARCHMAP","@value":"https://researchmap.jp/juntendo1227"}],"foaf:name":[{"@value":"Yusuke Suzuki"}]}],"publication":{"publicationIdentifier":[{"@type":"PISSN","@value":"00852538"}],"prism:publicationName":[{"@value":"Kidney International"}],"dc:publisher":[{"@value":"Elsevier BV"}],"prism:publicationDate":"2018-03","prism:volume":"93","prism:number":"3","prism:startingPage":"700","prism:endingPage":"705"},"reviewed":"false","dcterms:accessRights":"http://purl.org/coar/access_right/c_abf2","dc:rights":["https://www.elsevier.com/tdm/userlicense/1.0/","http://creativecommons.org/licenses/by-nc-nd/4.0/"],"url":[{"@id":"https://api.elsevier.com/content/article/PII:S0085253817307998?httpAccept=text/xml"},{"@id":"https://api.elsevier.com/content/article/PII:S0085253817307998?httpAccept=text/plain"}],"createdAt":"2018-01-09","modifiedAt":"2018-06-29","foaf:topic":[{"@id":"https://cir.nii.ac.jp/all?q=Vasculitis","dc:title":"Vasculitis"},{"@id":"https://cir.nii.ac.jp/all?q=Biopsy","dc:title":"Biopsy"},{"@id":"https://cir.nii.ac.jp/all?q=Kidney%20Glomerulus","dc:title":"Kidney Glomerulus"},{"@id":"https://cir.nii.ac.jp/all?q=Antibodies,%20Monoclonal","dc:title":"Antibodies, Monoclonal"},{"@id":"https://cir.nii.ac.jp/all?q=Fluorescent%20Antibody%20Technique","dc:title":"Fluorescent Antibody Technique"},{"@id":"https://cir.nii.ac.jp/all?q=Galactose","dc:title":"Galactose"},{"@id":"https://cir.nii.ac.jp/all?q=Glomerulonephritis,%20IGA","dc:title":"Glomerulonephritis, IGA"},{"@id":"https://cir.nii.ac.jp/all?q=Immunoglobulin%20A","dc:title":"Immunoglobulin A"},{"@id":"https://cir.nii.ac.jp/all?q=Microscopy,%20Fluorescence","dc:title":"Microscopy, Fluorescence"},{"@id":"https://cir.nii.ac.jp/all?q=Humans","dc:title":"Humans"}],"project":[{"@id":"https://cir.nii.ac.jp/crid/1040000781850743424","@type":"Project","projectIdentifier":[{"@type":"KAKEN","@value":"15K09274"},{"@type":"JGN","@value":"JP15K09274"},{"@type":"URI","@value":"https://kaken.nii.ac.jp/grant/KAKENHI-PROJECT-15K09274/"}],"notation":[{"@language":"ja","@value":"IgA腎症の病態におけるTLR/BAFF/APRILの役割の解明"},{"@language":"en","@value":"Role of TLR/BAFF/APRIL in the pathogenesis of IgA nephropathy"}]},{"@id":"https://cir.nii.ac.jp/crid/1040000781853156480","@type":"Project","projectIdentifier":[{"@type":"KAKEN","@value":"15K10643"},{"@type":"JGN","@value":"JP15K10643"},{"@type":"URI","@value":"https://kaken.nii.ac.jp/grant/KAKENHI-PROJECT-15K10643/"}],"notation":[{"@language":"ja","@value":"非侵襲的バイオマーカーによる腎移植後再発性腎炎の発症・活動性予測法の確立"},{"@language":"en","@value":"A single center prospective observational study for the purpose of non-invasive diagnosis of post-transplant IgA 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