{"@context":{"@vocab":"https://cir.nii.ac.jp/schema/1.0/","rdfs":"http://www.w3.org/2000/01/rdf-schema#","dc":"http://purl.org/dc/elements/1.1/","dcterms":"http://purl.org/dc/terms/","foaf":"http://xmlns.com/foaf/0.1/","prism":"http://prismstandard.org/namespaces/basic/2.0/","cinii":"http://ci.nii.ac.jp/ns/1.0/","datacite":"https://schema.datacite.org/meta/kernel-4/","ndl":"http://ndl.go.jp/dcndl/terms/","jpcoar":"https://github.com/JPCOAR/schema/blob/master/2.0/"},"@id":"https://cir.nii.ac.jp/crid/1362262945346162560.json","@type":"Article","productIdentifier":[{"identifier":{"@type":"DOI","@value":"10.1126/science.aaa2630"}},{"identifier":{"@type":"URI","@value":"https://www.science.org/doi/pdf/10.1126/science.aaa2630"}}],"dc:title":[{"@value":"Phosphorylation of innate immune adaptor proteins MAVS, STING, and TRIF induces IRF3 activation"}],"description":[{"type":"abstract","notation":[{"@value":"<jats:title>Innate immune receptor signaling, united</jats:title>\n          <jats:p>\n            Innate immune receptors such as RIG-I, cGAS, and Toll-like receptors bind microbial fragments and alert the immune system to an infection. Each receptor type signals through a different adapter protein. These signals activate the protein kinase TBK1 and the transcription factor IRF3, which tells cells to secrete interferon proteins (IFNs) important for host defense. Liu\n            <jats:italic>et al.</jats:italic>\n            now report a common signaling mechanism used by all three types of innate immune receptor-adaptor protein pairs to activate IRF3 and generate IFNs. This is important because cells must regulate their IFN production carefully to avoid inflammation and autoimmunity.\n          </jats:p>\n          <jats:p>\n            <jats:italic>Science</jats:italic>\n            , this issue\n            <jats:related-article xmlns:xlink=\"http://www.w3.org/1999/xlink\" ext-link-type=\"doi\" related-article-type=\"in-this-issue\" xlink:href=\"10.1126/science.aaa2630\">10.1126/science.aaa2630</jats:related-article>\n          </jats:p>"}]}],"creator":[{"@id":"https://cir.nii.ac.jp/crid/1382262945346162689","@type":"Researcher","foaf:name":[{"@value":"Siqi Liu"}],"jpcoar:affiliationName":[{"@value":"Department of Molecular Biology, University of Texas Southwestern Medical Center, Dallas, TX 75390-9148, USA."}]},{"@id":"https://cir.nii.ac.jp/crid/1382262945346162560","@type":"Researcher","foaf:name":[{"@value":"Xin Cai"}],"jpcoar:affiliationName":[{"@value":"Department of Molecular Biology, University of Texas Southwestern Medical Center, Dallas, TX 75390-9148, USA."}]},{"@id":"https://cir.nii.ac.jp/crid/1382262945346162561","@type":"Researcher","foaf:name":[{"@value":"Jiaxi Wu"}],"jpcoar:affiliationName":[{"@value":"Department of Molecular Biology, University of Texas Southwestern Medical Center, Dallas, TX 75390-9148, USA."}]},{"@id":"https://cir.nii.ac.jp/crid/1382262945346162562","@type":"Researcher","foaf:name":[{"@value":"Qian Cong"}],"jpcoar:affiliationName":[{"@value":"Departments of Biophysics and Biochemistry, University of Texas Southwestern Medical Center, Dallas, TX 75390-9148, USA."}]},{"@id":"https://cir.nii.ac.jp/crid/1382262945346162565","@type":"Researcher","foaf:name":[{"@value":"Xiang Chen"}],"jpcoar:affiliationName":[{"@value":"Department of Molecular Biology, University of Texas Southwestern Medical Center, Dallas, TX 75390-9148, USA."},{"@value":"Howard Hughes Medical Institute (HHMI), University of Texas Southwestern Medical Center, Dallas, TX 75390-9148, USA."}]},{"@id":"https://cir.nii.ac.jp/crid/1382262945346162564","@type":"Researcher","foaf:name":[{"@value":"Tuo Li"}],"jpcoar:affiliationName":[{"@value":"Department of Molecular Biology, University of Texas Southwestern Medical Center, Dallas, TX 75390-9148, USA."}]},{"@id":"https://cir.nii.ac.jp/crid/1382262945346162567","@type":"Researcher","foaf:name":[{"@value":"Fenghe Du"}],"jpcoar:affiliationName":[{"@value":"Department of Molecular Biology, University of Texas Southwestern Medical Center, Dallas, TX 75390-9148, USA."},{"@value":"Howard Hughes Medical Institute (HHMI), University of Texas Southwestern Medical Center, Dallas, TX 75390-9148, USA."}]},{"@id":"https://cir.nii.ac.jp/crid/1382262945346162688","@type":"Researcher","foaf:name":[{"@value":"Junyao Ren"}],"jpcoar:affiliationName":[{"@value":"Department of Molecular Biology, University of Texas Southwestern Medical Center, Dallas, TX 75390-9148, USA."}]},{"@id":"https://cir.nii.ac.jp/crid/1382262945346162568","@type":"Researcher","foaf:name":[{"@value":"You-Tong Wu"}],"jpcoar:affiliationName":[{"@value":"Department of Molecular Biology, University of Texas Southwestern Medical Center, Dallas, TX 75390-9148, USA."}]},{"@id":"https://cir.nii.ac.jp/crid/1382262945346162563","@type":"Researcher","foaf:name":[{"@value":"Nick V. Grishin"}],"jpcoar:affiliationName":[{"@value":"Departments of Biophysics and Biochemistry, University of Texas Southwestern Medical Center, Dallas, TX 75390-9148, USA."},{"@value":"Howard Hughes Medical Institute (HHMI), University of Texas Southwestern Medical Center, Dallas, TX 75390-9148, USA."}]},{"@id":"https://cir.nii.ac.jp/crid/1382262945346162566","@type":"Researcher","foaf:name":[{"@value":"Zhijian J. Chen"}],"jpcoar:affiliationName":[{"@value":"Department of Molecular Biology, University of Texas Southwestern Medical Center, Dallas, TX 75390-9148, USA."},{"@value":"Howard Hughes Medical Institute (HHMI), University of Texas Southwestern Medical Center, Dallas, TX 75390-9148, USA."}]}],"publication":{"publicationIdentifier":[{"@type":"PISSN","@value":"00368075"},{"@type":"EISSN","@value":"10959203"}],"prism:publicationName":[{"@value":"Science"}],"dc:publisher":[{"@value":"American Association for the Advancement of Science (AAAS)"}],"prism:publicationDate":"2015-03-13","prism:volume":"347","prism:number":"6227","prism:startingPage":"aaa2630"},"reviewed":"false","dc:rights":["http://www.sciencemag.org/about/science-licenses-journal-article-reuse"],"url":[{"@id":"https://www.science.org/doi/pdf/10.1126/science.aaa2630"}],"createdAt":"2015-01-30","modifiedAt":"2024-01-15","relatedProduct":[{"@id":"https://cir.nii.ac.jp/crid/1050017655056227200","@type":"Article","resourceType":"学術雑誌論文(journal article)","relationType":["isReferencedBy"],"jpcoar:relatedTitle":[{"@language":"en","@value":"Micronucleus is not a potent inducer of the cGAS/STING pathway"}]},{"@id":"https://cir.nii.ac.jp/crid/1050282677611262464","@type":"Article","resourceType":"学術雑誌論文(journal article)","relationType":["isReferencedBy"],"jpcoar:relatedTitle":[{"@language":"en","@value":"The RAB2B-GARIL5 Complex Promotes Cytosolic DNA-Induced Innate Immune Responses"},{"@value":"The RAB2B-GARIL5 complex promotes cytosolic dna-induced innate immune responses article the RAB2B-GARIL5 complex promotes cytosolic dna-induced innate immune responses"}]},{"@id":"https://cir.nii.ac.jp/crid/1050295834376031360","@type":"Article","resourceType":"レビュー論文(review article)","relationType":["isReferencedBy"],"jpcoar:relatedTitle":[{"@language":"en","@value":"Pathophysiological functions of self-derived DNA"}]},{"@id":"https://cir.nii.ac.jp/crid/1050866647413161984","@type":"Article","resourceType":"学術雑誌論文(journal article)","relationType":["isReferencedBy"],"jpcoar:relatedTitle":[{"@language":"en","@value":"Receptor-activated transcription factors and beyond: multiple modes of Smad2/3-dependent transmission of TGF-β signaling"}]},{"@id":"https://cir.nii.ac.jp/crid/1050867133848389632","@type":"Article","resourceType":"学術雑誌論文(journal article)","relationType":["isReferencedBy"],"jpcoar:relatedTitle":[{"@language":"en","@value":"The Golgi-resident protein ACBD3 concentrates STING at ER-Golgi contact sites to drive export from the 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Carcinoma"}]},{"@id":"https://cir.nii.ac.jp/crid/1360306904385462144","@type":"Article","resourceType":"学術雑誌論文(journal article)","relationType":["isReferencedBy"],"jpcoar:relatedTitle":[{"@value":"Heme regulates protein interactions and phosphorylation of BACH2 intrinsically disordered region in humoral response"}]},{"@id":"https://cir.nii.ac.jp/crid/1360306905165662208","@type":"Article","resourceType":"学術雑誌論文(journal article)","relationType":["isReferencedBy"],"jpcoar:relatedTitle":[{"@value":"ArfGAP2 promotes STING proton channel activity, cytokine transit, and autoinflammation"}]},{"@id":"https://cir.nii.ac.jp/crid/1360565167103179264","@type":"Article","resourceType":"学術雑誌論文(journal article)","relationType":["isReferencedBy"],"jpcoar:relatedTitle":[{"@value":"Phosphoproteomics to Characterize Host Response During Influenza A Virus Infection of Human Macrophages"}]},{"@id":"https://cir.nii.ac.jp/crid/1360565168590683648","@type":"Article","resourceType":"学術雑誌論文(journal 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ER"}]},{"@id":"https://cir.nii.ac.jp/crid/1360855570650432000","@type":"Article","resourceType":"preprint","relationType":["isReferencedBy"],"jpcoar:relatedTitle":[{"@value":"Homeostatic regulation of STING by Golgi-to-ER membrane traffic"}]},{"@id":"https://cir.nii.ac.jp/crid/1360861705595997440","@type":"Article","resourceType":"学術雑誌論文(journal article)","relationType":["isReferencedBy"],"jpcoar:relatedTitle":[{"@value":"Exposure to microbial products followed by loss of Tet2 promotes myelodysplastic syndrome via remodeling HSCs"}]},{"@id":"https://cir.nii.ac.jp/crid/1360861707161921792","@type":"Article","resourceType":"学術雑誌論文(journal article)","relationType":["isReferencedBy"],"jpcoar:relatedTitle":[{"@value":"Telomere Maintenance and the cGAS-STING Pathway in Cancer"}]},{"@id":"https://cir.nii.ac.jp/crid/1360865814738904448","@type":"Article","resourceType":"学術雑誌論文(journal article)","relationType":["isReferencedBy"],"jpcoar:relatedTitle":[{"@value":"The tumor cell-intrinsic 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