{"@context":{"@vocab":"https://cir.nii.ac.jp/schema/1.0/","rdfs":"http://www.w3.org/2000/01/rdf-schema#","dc":"http://purl.org/dc/elements/1.1/","dcterms":"http://purl.org/dc/terms/","foaf":"http://xmlns.com/foaf/0.1/","prism":"http://prismstandard.org/namespaces/basic/2.0/","cinii":"http://ci.nii.ac.jp/ns/1.0/","datacite":"https://schema.datacite.org/meta/kernel-4/","ndl":"http://ndl.go.jp/dcndl/terms/","jpcoar":"https://github.com/JPCOAR/schema/blob/master/2.0/"},"@id":"https://cir.nii.ac.jp/crid/1363388844091722752.json","@type":"Article","productIdentifier":[{"identifier":{"@type":"DOI","@value":"10.1016/s1535-6108(04)00021-2"}},{"identifier":{"@type":"URI","@value":"https://api.elsevier.com/content/article/PII:S1535610804000212?httpAccept=text/xml"}},{"identifier":{"@type":"URI","@value":"https://api.elsevier.com/content/article/PII:S1535610804000212?httpAccept=text/plain"}}],"dc:title":[{"@value":"Prune cAMP phosphodiesterase binds nm23-H1 and promotes cancer metastasis"}],"creator":[{"@id":"https://cir.nii.ac.jp/crid/1383388844091722752","@type":"Researcher","foaf:name":[{"@value":"Anna D'Angelo"}]},{"@id":"https://cir.nii.ac.jp/crid/1383388844091722754","@type":"Researcher","foaf:name":[{"@value":"Livia Garzia"}]},{"@id":"https://cir.nii.ac.jp/crid/1383388844091722753","@type":"Researcher","foaf:name":[{"@value":"Alessandra André"}]},{"@id":"https://cir.nii.ac.jp/crid/1383388844091722762","@type":"Researcher","foaf:name":[{"@value":"Pietro Carotenuto"}]},{"@id":"https://cir.nii.ac.jp/crid/1383388844091722759","@type":"Researcher","foaf:name":[{"@value":"Veruska Aglio"}]},{"@id":"https://cir.nii.ac.jp/crid/1383388844091722760","@type":"Researcher","foaf:name":[{"@value":"Ombretta Guardiola"}]},{"@id":"https://cir.nii.ac.jp/crid/1383388844091722755","@type":"Researcher","foaf:name":[{"@value":"Gianluigi Arrigoni"}]},{"@id":"https://cir.nii.ac.jp/crid/1383388844091722756","@type":"Researcher","foaf:name":[{"@value":"Antonio Cossu"}]},{"@id":"https://cir.nii.ac.jp/crid/1383388844091722761","@type":"Researcher","foaf:name":[{"@value":"Giuseppe Palmieri"}]},{"@id":"https://cir.nii.ac.jp/crid/1383388844091722757","@type":"Researcher","foaf:name":[{"@value":"L Aravind"}]},{"@id":"https://cir.nii.ac.jp/crid/1383388844091722758","@type":"Researcher","foaf:name":[{"@value":"Massimo Zollo"}]}],"publication":{"publicationIdentifier":[{"@type":"PISSN","@value":"15356108"}],"prism:publicationName":[{"@value":"Cancer Cell"}],"dc:publisher":[{"@value":"Elsevier BV"}],"prism:publicationDate":"2004-02","prism:volume":"5","prism:number":"2","prism:startingPage":"137","prism:endingPage":"149"},"reviewed":"false","dc:rights":["https://www.elsevier.com/tdm/userlicense/1.0/","https://www.elsevier.com/open-access/userlicense/1.0/"],"url":[{"@id":"https://api.elsevier.com/content/article/PII:S1535610804000212?httpAccept=text/xml"},{"@id":"https://api.elsevier.com/content/article/PII:S1535610804000212?httpAccept=text/plain"}],"createdAt":"2004-04-02","modifiedAt":"2021-06-15","relatedProduct":[{"@id":"https://cir.nii.ac.jp/crid/1360004235494902656","@type":"Article","resourceType":"学術雑誌論文(journal article)","relationType":["isReferencedBy"],"jpcoar:relatedTitle":[{"@value":"<i>PRUNE1</i>‐related disorder: Expanding the clinical spectrum"}]},{"@id":"https://cir.nii.ac.jp/crid/1360283693644273536","@type":"Article","resourceType":"学術雑誌論文(journal article)","relationType":["isReferencedBy"],"jpcoar:relatedTitle":[{"@value":"Extracellular NM23 Protein as a Therapeutic Target for Hematologic Malignancies"}]},{"@id":"https://cir.nii.ac.jp/crid/1360285714671287808","@type":"Article","resourceType":"学術雑誌論文(journal article)","relationType":["isReferencedBy"],"jpcoar:relatedTitle":[{"@value":"h-prune affects anaplastic thyroid cancer invasion and metastasis"}]},{"@id":"https://cir.nii.ac.jp/crid/1360567180092274816","@type":"Article","resourceType":"学術雑誌論文(journal article)","relationType":["isReferencedBy"],"jpcoar:relatedTitle":[{"@value":"h‐Prune is associated with poor prognosis and epithelial–mesenchymal transition in patients with colorectal liver metastases"}]},{"@id":"https://cir.nii.ac.jp/crid/1360567185505575296","@type":"Article","resourceType":"学術雑誌論文(journal article)","relationType":["isReferencedBy"],"jpcoar:relatedTitle":[{"@value":"DNA glycosylase encoded by <i>MUTYH</i> functions as a molecular switch for programmed cell death under oxidative stress to suppress tumorigenesis"}]}],"dataSourceIdentifier":[{"@type":"CROSSREF","@value":"10.1016/s1535-6108(04)00021-2"},{"@type":"CROSSREF","@value":"10.1111/cge.13385_references_DOI_QOSwsRVyuLMKNvzxUJzJGgIZcZh"},{"@type":"CROSSREF","@value":"10.3892/or.2016.4759_references_DOI_QOSwsRVyuLMKNvzxUJzJGgIZcZh"},{"@type":"CROSSREF","@value":"10.1155/2012/879368_references_DOI_QOSwsRVyuLMKNvzxUJzJGgIZcZh"},{"@type":"CROSSREF","@value":"10.1002/ijc.30118_references_DOI_QOSwsRVyuLMKNvzxUJzJGgIZcZh"},{"@type":"CROSSREF","@value":"10.1007/s12031-010-9490-2_references_DOI_QOSwsRVyuLMKNvzxUJzJGgIZcZh"}]}