{"@context":{"@vocab":"https://cir.nii.ac.jp/schema/1.0/","rdfs":"http://www.w3.org/2000/01/rdf-schema#","dc":"http://purl.org/dc/elements/1.1/","dcterms":"http://purl.org/dc/terms/","foaf":"http://xmlns.com/foaf/0.1/","prism":"http://prismstandard.org/namespaces/basic/2.0/","cinii":"http://ci.nii.ac.jp/ns/1.0/","datacite":"https://schema.datacite.org/meta/kernel-4/","ndl":"http://ndl.go.jp/dcndl/terms/","jpcoar":"https://github.com/JPCOAR/schema/blob/master/2.0/"},"@id":"https://cir.nii.ac.jp/crid/1363670318300575232.json","@type":"Article","productIdentifier":[{"identifier":{"@type":"DOI","@value":"10.1161/01.cir.96.6.1859"}},{"identifier":{"@type":"URI","@value":"https://www.ahajournals.org/doi/full/10.1161/01.CIR.96.6.1859"}}],"dc:title":[{"@value":"Sodium Restriction Shifts Circadian Rhythm of Blood Pressure From Nondipper to Dipper in Essential Hypertension"}],"description":[{"type":"abstract","notation":[{"@value":"<jats:p>\n            <jats:italic>Background</jats:italic>\n            Sodium restriction has been widely used for treatment of hypertension and renal diseases. Whether sodium restriction can transform the circadian rhythm of blood pressure from nondipper to dipper is examined.\n          </jats:p>\n          <jats:p>\n            <jats:italic>Methods and Results</jats:italic>\n            Patients (n=42) with essential hypertension were maintained on a high-sodium diet (12 to 15 g of NaCl per day) and a low-sodium diet (1 to 3 g/d) for 1 week each. On the last day of each diet, blood pressures were measured noninvasively every hour for 24 hours with an automatic oscillometric device. Twenty-one patients were classified as non–sodium sensitive whereas 21 were classified as sodium sensitive on the basis of a ≥10% change in 24-hour mean arterial pressure caused by sodium restriction. Nocturnal blood pressure fall was significant in the non–sodium sensitive subjects but not in sodium-sensitive subjects. There was a significant interaction between sodium restriction and nocturnal fall in blood pressure only in the sodium-sensitive subjects, indicating that the degree of the nocturnal fall was affected by sodium restriction. Furthermore, changes in the nocturnal fall induced by sodium restriction had a positive relationship with sodium sensitivity (\n            <jats:italic>r</jats:italic>\n            =.38,\n            <jats:italic>P</jats:italic>\n            <.02) and a negative relationship with the nocturnal fall before sodium restriction (\n            <jats:italic>r</jats:italic>\n            =−.75,\n            <jats:italic>P</jats:italic>\n            <.0001).\n          </jats:p>\n          <jats:p>\n            <jats:italic>Conclusions</jats:italic>\n            These findings show the difference in nocturnal fall in blood pressure between the non–sodium sensitive and sodium-sensitive types of essential hypertension. The diminished nocturnal fall, recognized in the sodium-sensitive type, is restored by sodium restriction, indicating that the circadian rhythm of blood pressure shifted from a nondipper to a dipper pattern. On the other hand, the nocturnal fall is not affected by sodium restriction in the non–sodium sensitive type, and the circadian rhythm remains of the dipper variety.\n          </jats:p>"}]}],"creator":[{"@id":"https://cir.nii.ac.jp/crid/1383670318300575233","@type":"Researcher","foaf:name":[{"@value":"Takashi Uzu"}],"jpcoar:affiliationName":[{"@value":"From the Division of Nephrology, National Cardiovascular Center, Osaka, Japan."}]},{"@id":"https://cir.nii.ac.jp/crid/1383670318300575236","@type":"Researcher","foaf:name":[{"@value":"Kazuhiko Ishikawa"}],"jpcoar:affiliationName":[{"@value":"From the Division of Nephrology, National Cardiovascular Center, Osaka, Japan."}]},{"@id":"https://cir.nii.ac.jp/crid/1383670318300575232","@type":"Researcher","foaf:name":[{"@value":"Takashi Fujii"}],"jpcoar:affiliationName":[{"@value":"From the Division of Nephrology, National Cardiovascular Center, Osaka, Japan."}]},{"@id":"https://cir.nii.ac.jp/crid/1383670318300575234","@type":"Researcher","foaf:name":[{"@value":"Satoko Nakamura"}],"jpcoar:affiliationName":[{"@value":"From the Division of Nephrology, National Cardiovascular Center, Osaka, Japan."}]},{"@id":"https://cir.nii.ac.jp/crid/1383670318300575235","@type":"Researcher","foaf:name":[{"@value":"Takashi Inenaga"}],"jpcoar:affiliationName":[{"@value":"From the Division of Nephrology, National Cardiovascular Center, Osaka, Japan."}]},{"@id":"https://cir.nii.ac.jp/crid/1383670318300575237","@type":"Researcher","foaf:name":[{"@value":"Genjiro Kimura"}],"jpcoar:affiliationName":[{"@value":"From the Division of Nephrology, National Cardiovascular Center, Osaka, Japan."}]}],"publication":{"publicationIdentifier":[{"@type":"PISSN","@value":"00097322"},{"@type":"EISSN","@value":"15244539"},{"@type":"PISSN","@value":"http://id.crossref.org/issn/00097322"},{"@type":"PISSN","@value":"https://id.crossref.org/issn/00097322"}],"prism:publicationName":[{"@value":"Circulation"}],"dc:publisher":[{"@value":"Ovid Technologies (Wolters Kluwer Health)"}],"prism:publicationDate":"1997-09-16","prism:volume":"96","prism:number":"6","prism:startingPage":"1859","prism:endingPage":"1862"},"reviewed":"false","url":[{"@id":"https://www.ahajournals.org/doi/full/10.1161/01.CIR.96.6.1859"}],"createdAt":"2012-06-12","modifiedAt":"2024-05-12","relatedProduct":[{"@id":"https://cir.nii.ac.jp/crid/1360004234890171648","@type":"Article","resourceType":"学術雑誌論文(journal article)","relationType":["isReferencedBy"],"jpcoar:relatedTitle":[{"@value":"B-type natriuretic peptide is a determinant of the nocturnal increase in blood pressure independently of arterial hypertrophy and 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