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Dystrophin defect affects cardiac muscle as well as skeletal muscle. Cardiac dysfunction is observed in all patients with DMD over 18 years of age, but there is no curative treatment for DMD cardiomyopathy. To establish novel experimental platforms which reproduce the cardiac phenotype of DMD patients, here we established iPS cell lines from T lymphocytes donated from two DMD patients, with a protocol using Sendai virus vectors. We successfully conducted the differentiation of the DMD patient-specific iPS cells into beating cardiomyocytes. DMD patient-specific iPS cells and iPS cell-derived cardiomyocytes would be a useful <i>in vitro</i> experimental system with which to investigate DMD cardiomyopathy."}],"abstractLicenseFlag":"disallow"}],"creator":[{"@id":"https://cir.nii.ac.jp/crid/1410001205226540040","@type":"Researcher","personIdentifier":[{"@type":"NRID","@value":"9000314067571"}],"foaf:name":[{"@language":"en","@value":"Hashimoto Akihito"}],"jpcoar:affiliationName":[{"@language":"en","@value":"Department of Cardiovascular Regenerative Medicine, Osaka University Graduate School of Medicine"},{"@language":"en","@value":"Department of Cardiovascular Medicine, Osaka University Graduate School of Medicine"}]},{"@id":"https://cir.nii.ac.jp/crid/1410001205226540037","@type":"Researcher","personIdentifier":[{"@type":"NRID","@value":"9000314067592"}],"foaf:name":[{"@language":"en","@value":"Sumida Tomokazu"}],"jpcoar:affiliationName":[{"@language":"en","@value":"Department of Cardiovascular Medicine, Graduate School of 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