Pancreatic injury in primary biliary cirrhosis.

DOI PubMed
  • KIDANI Hisashi
    The First Department of Internal Medicine, Kanazawa University The Department of Internal Medicine, Cancer Institute, Kanazawa University
  • SAWABU Norio
    The First Department of Internal Medicine, Kanazawa University The Department of Internal Medicine, Cancer Institute, Kanazawa University
  • TOYA Daishu
    The First Department of Internal Medicine, Kanazawa University
  • YONEJIMA Masahiro
    The First Department of Internal Medicine, Kanazawa University
  • TAKEMORI Yasuhiro
    The First Department of Internal Medicine, Kanazawa University
  • YONEJIMA Manabu
    The First Department of Internal Medicine, Kanazawa University The Department of Internal Medicine, Cancer Institute, Kanazawa University
  • MIYAMORI Hirotoshi
    The First Department of Internal Medicine, Kanazawa University
  • WAKABAYASHI Tokio
    The First Department of Internal Medicine, Kanazawa University
  • OZAKI Kenji
    The First Department of Internal Medicine, Kanazawa University
  • KATO Yasuhiro
    The First Department of Internal Medicine, Kanazawa University
  • KOBAYASHI Kenichi
    The First Department of Internal Medicine, Kanazawa University
  • HATTORI Nobu
    The First Department of Internal Medicine, Kanazawa University
  • NAKANUMA Yasuni
    The Second Department of Pathology, School of Medicine, Kanazawa University

Bibliographic Information

Other Title
  • 原発性胆汁性肝硬変におけるすい病変について
Published
1984
Resource Type
journal article
DOI
  • 10.11405/nisshoshi1964.81.2544
Publisher
The Japanese Society of Gastroenterology

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Description

The present study was undertaken to clarify the functional and/or morphological pancreatic injury in patients with primary biliary cirrhosis (PBC).<br>Clinical observation of the pancreas was done on 22 patients with PBC chiefly on the basis of pancreozyminsecretin (PS) test and endoscopic retrograde pancreatocholangiography (ERCP). In addition, 4 autopsied pancreases from patients with PBC were examined pathologically. PS tests were abnormal in 27% of 15 cases and ERCP were abnormal in 47% of 17 cases. However, these changes remained mild except for one patient. Abnormalities of PS test or ERCP were not correlated with the pathological stages of PBC or the association of sicca complex. In 2 of 4 autopsied pancreases, findings of mild chronic pancreatitis with lymphocytic infiltration were observed. These results suggest that pancreatic injury may occur with considerable frequency in PBC, but this injury is almost always slight.

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