{"@context":{"@vocab":"https://cir.nii.ac.jp/schema/1.0/","rdfs":"http://www.w3.org/2000/01/rdf-schema#","dc":"http://purl.org/dc/elements/1.1/","dcterms":"http://purl.org/dc/terms/","foaf":"http://xmlns.com/foaf/0.1/","prism":"http://prismstandard.org/namespaces/basic/2.0/","cinii":"http://ci.nii.ac.jp/ns/1.0/","datacite":"https://schema.datacite.org/meta/kernel-4/","ndl":"http://ndl.go.jp/dcndl/terms/","jpcoar":"https://github.com/JPCOAR/schema/blob/master/2.0/"},"@id":"https://cir.nii.ac.jp/crid/1871709542852817536.json","@type":"Article","productIdentifier":[{"identifier":{"@type":"DOI","@value":"10.1016/s0016-5085(76)80307-1"}},{"identifier":{"@type":"PMID","@value":"1269863"}}],"dc:title":[{"@value":"Tissue Plasminogen Activator in the Pathogenesis of Protein-Losing Gastroenteropathy"}],"description":[{"notation":[{"@value":"Tissue fibrinolytic activity, mainly due to activator of tissue plasminogen was increased in the biopsied gastric mucosa of patients with protein-losing gastropathy. Antiplasmin therapy with trans-AMCHA (trans-4-aminomethyl cyclohexane carboxylic acid) was dramatically effective, except in the case of a patient with intestinal lymphangiectasia. These findings suggest that increased fibrinolysis in the gastrointestinal mucosa may play an important role in enhancing mucosal permeability to plasma proteins."}]}],"creator":[{"@id":"https://cir.nii.ac.jp/crid/1891709542852817541","@type":"Researcher","foaf:name":[{"@value":"Tadao Bamba"}]},{"@id":"https://cir.nii.ac.jp/crid/1891709542852817539","@type":"Researcher","foaf:name":[{"@value":"Keimei Hosokawa"}]},{"@id":"https://cir.nii.ac.jp/crid/1891709542852817536","@type":"Researcher","foaf:name":[{"@value":"S. Hosoda"}]},{"@id":"https://cir.nii.ac.jp/crid/1891709542852817537","@type":"Researcher","foaf:name":[{"@value":"Masasuke Masuda"}]},{"@id":"https://cir.nii.ac.jp/crid/1891709542852817538","@type":"Researcher","foaf:name":[{"@value":"Keiichi Kawai"}]},{"@id":"https://cir.nii.ac.jp/crid/1891709542852817540","@type":"Researcher","foaf:name":[{"@value":"Motoharu Kondo"}]}],"publication":{"publicationIdentifier":[{"@type":"PISSN","@value":"00165085"}],"prism:publicationName":[{"@value":"Gastroenterology"}],"dc:publisher":[{"@value":"Elsevier BV"}],"prism:publicationDate":"1976-06-01","prism:volume":"70","prism:startingPage":"1045","prism:endingPage":"1047"},"dcterms:accessRights":"http://purl.org/coar/access_right/c_abf2","foaf:topic":[{"@id":"https://cir.nii.ac.jp/all?q=Adult","dc:title":"Adult"},{"@id":"https://cir.nii.ac.jp/all?q=Male","dc:title":"Male"},{"@id":"https://cir.nii.ac.jp/all?q=Plasminogen%20Activators","dc:title":"Plasminogen Activators"},{"@id":"https://cir.nii.ac.jp/all?q=Tranexamic%20Acid","dc:title":"Tranexamic Acid"},{"@id":"https://cir.nii.ac.jp/all?q=Gastric%20Mucosa","dc:title":"Gastric Mucosa"},{"@id":"https://cir.nii.ac.jp/all?q=Fibrinolysis","dc:title":"Fibrinolysis"},{"@id":"https://cir.nii.ac.jp/all?q=Protein-Losing%20Enteropathies","dc:title":"Protein-Losing Enteropathies"},{"@id":"https://cir.nii.ac.jp/all?q=Humans","dc:title":"Humans"}],"dataSourceIdentifier":[{"@type":"OPENAIRE","@value":"doi_dedup___::a84be6ac862d5fe078233e63043a6942"}]}