Establishment of in vivo and in vitro systems to study the pathophysiology of disuse muscle atrophy

About This Project

Japan Grant Number
JP18K19747 (JGN)
Funding Program
Grants-in-Aid for Scientific Research
Funding Organization
Japan Society for the Promotion of Science

Kakenhi Information

Project/Area Number
18K19747
Research Category
Grant-in-Aid for Challenging Research (Exploratory)
Allocation Type
  • Multi-year Fund
Review Section / Research Field
  • Medium-sized Section 59:Sports sciences, physical education, health sciences, and related fields
Research Institution
  • Kyushu University
Project Period (FY)
2018-06-29 〜 2022-03-31
Project Status
Completed
Budget Amount*help
6,500,000 Yen (Direct Cost: 5,000,000 Yen Indirect Cost: 1,500,000 Yen)

Research Abstract

We aim to clarify the pathological role of the oxidative stress in disuse muscle atrophy from a cell biological point of view. We previously observed the fragmentation of the Golgi apparatus in atrophied cells, which suggesting the disfunction of the intracellular transport system. One possible mechanism of the Golgi fragmentation is the inhibition of membrane fusion. In this study, we focused on a membrane tethering machinery required for the membrane fusion, especially for its specificity, in the Golgi. We have identified a novel membrane tethering factor, FTCD, in the Golgi and have succeeded in establishing both in vivo and in vitro systems which enable us to monitor the assembly/disassembly of the Golgi membrane tethering machinery in living cells and a test tube.

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