Two guard cell mitogen‐activated protein kinases, <scp>MPK</scp>9 and <scp>MPK</scp>12, function in methyl jasmonate‐induced stomatal closure in <i>Arabidopsis thaliana</i>

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  • Md. A. R. Khokon
    Graduate School of Environmental and Life Science Okayama University Okayama Japan
  • M. A. Salam
    Graduate School of Environmental and Life Science Okayama University Okayama Japan
  • F. Jammes
    Department of Biology Pomona College Claremont CA USA
  • W. Ye
    Graduate School of Environmental and Life Science Okayama University Okayama Japan
  • M. A. Hossain
    Graduate School of Environmental and Life Science Okayama University Okayama Japan
  • M. Uraji
    Graduate School of Environmental and Life Science Okayama University Okayama Japan
  • Y. Nakamura
    Graduate School of Environmental and Life Science Okayama University Okayama Japan
  • I. C. Mori
    Institute of Plant Science and Resources Okayama University Kurashiki Okayama Japan
  • J. M. Kwak
    Department of New Biology Center for Plant Aging Research Institute for Basic Science Daegu Kyungbuk Institute of Science and Technology Daegu Korea
  • Y. Murata
    Graduate School of Environmental and Life Science Okayama University Okayama Japan

書誌事項

公開日
2015-03-17
資源種別
journal article
権利情報
  • http://onlinelibrary.wiley.com/termsAndConditions#vor
DOI
  • 10.1111/plb.12321
公開者
Wiley

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説明

<jats:title>Abstract</jats:title><jats:p>Methyl jasmonate (Me<jats:styled-content style="fixed-case">JA</jats:styled-content>) and abscisic acid (<jats:styled-content style="fixed-case">ABA</jats:styled-content>) signalling cascades share several signalling components in guard cells. We previously showed that two guard cell‐preferential mitogen‐activated protein kinases (<jats:styled-content style="fixed-case">MAPK</jats:styled-content>s), <jats:italic><jats:styled-content style="fixed-case">MPK</jats:styled-content>9</jats:italic> and <jats:italic><jats:styled-content style="fixed-case">MPK</jats:styled-content>12</jats:italic>, positively regulate <jats:styled-content style="fixed-case">ABA</jats:styled-content> signalling in <jats:italic>Arabidopsis thaliana</jats:italic>. In this study, we examined whether these two <jats:styled-content style="fixed-case">MAP</jats:styled-content> kinases function in Me<jats:styled-content style="fixed-case">JA</jats:styled-content> signalling using genetic mutants for <jats:italic><jats:styled-content style="fixed-case">MPK</jats:styled-content>9</jats:italic> and <jats:italic><jats:styled-content style="fixed-case">MPK</jats:styled-content>12</jats:italic> combined with a pharmacological approach. Me<jats:styled-content style="fixed-case">JA</jats:styled-content> induced stomatal closure in <jats:italic>mpk9‐1</jats:italic> and <jats:italic>mpk12‐1</jats:italic> single mutants as well as wild‐type plants, but not in <jats:italic>mpk9‐1 mpk12‐1</jats:italic> double mutants. Consistently, the <jats:styled-content style="fixed-case">MAPKK</jats:styled-content> inhibitor <jats:styled-content style="fixed-case">PD</jats:styled-content>98059 inhibited the Me<jats:styled-content style="fixed-case">JA</jats:styled-content>‐induced stomatal closure in wild‐type plants. Me<jats:styled-content style="fixed-case">JA</jats:styled-content> elicited reactive oxygen species (<jats:styled-content style="fixed-case">ROS</jats:styled-content>) production and cytosolic alkalisation in guard cells of the <jats:italic>mpk9‐1, mpk12‐1</jats:italic> and <jats:italic>mpk9‐1 mpk12‐1</jats:italic> mutants, as well in wild‐type plants. Furthermore, Me<jats:styled-content style="fixed-case">JA</jats:styled-content> triggered elevation of cytosolic Ca<jats:sup>2+</jats:sup> concentration ([Ca<jats:sup>2+</jats:sup>]<jats:sub>cyt</jats:sub>) in the <jats:italic>mpk9‐1 mpk12‐1</jats:italic> double mutant as well as wild‐type plants. Activation of S‐type anion channels by Me<jats:styled-content style="fixed-case">JA</jats:styled-content> was impaired in <jats:italic>mpk9‐1 mpk12‐1</jats:italic>. Together, these results indicate that <jats:styled-content style="fixed-case">MPK</jats:styled-content>9 and <jats:styled-content style="fixed-case">MPK</jats:styled-content>12 function upstream of S‐type anion channel activation and downstream of <jats:styled-content style="fixed-case">ROS</jats:styled-content> production, cytosolic alkalisation and [Ca<jats:sup>2+</jats:sup>]<jats:sub>cyt</jats:sub> elevation in guard cell Me<jats:styled-content style="fixed-case">JA</jats:styled-content> signalling, suggesting that <jats:styled-content style="fixed-case">MPK</jats:styled-content>9 and <jats:styled-content style="fixed-case">MPK</jats:styled-content>12 are key regulators mediating both <jats:styled-content style="fixed-case">ABA</jats:styled-content> and Me<jats:styled-content style="fixed-case">JA</jats:styled-content> signalling in guard cells.</jats:p>

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