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- Dai Fujikawa
- Graduate School of Frontier Sciences, Department of Computational Biology and Medical Sciences, and
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- Shota Nakagawa
- Graduate School of Frontier Sciences, Department of Computational Biology and Medical Sciences, and
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- Makoto Hori
- Graduate School of Frontier Sciences, Department of Computational Biology and Medical Sciences, and
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- Naoya Kurokawa
- Graduate School of Frontier Sciences, Department of Computational Biology and Medical Sciences, and
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- Ai Soejima
- Graduate School of Frontier Sciences, Department of Computational Biology and Medical Sciences, and
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- Kazumi Nakano
- Graduate School of Frontier Sciences, Department of Computational Biology and Medical Sciences, and
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- Tadanori Yamochi
- Graduate School of Frontier Sciences, Department of Computational Biology and Medical Sciences, and
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- Makoto Nakashima
- Graduate School of Frontier Sciences, Department of Computational Biology and Medical Sciences, and
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- Seiichiro Kobayashi
- Institute of Medical Science, The University of Tokyo, Tokyo, Japan;
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- Yuetsu Tanaka
- Graduate School and Faculty of Medicine, University of the Ryukyus, Okinawa, Japan;
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- Masako Iwanaga
- Graduate School of Biomedical Sciences, Nagasaki University, Nagasaki, Japan; and
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- Atae Utsunomiya
- Department of Hematology, Imamura Bun-in Hospital, Kagoshima, Japan
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- Kaoru Uchimaru
- Institute of Medical Science, The University of Tokyo, Tokyo, Japan;
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- Makoto Yamagishi
- Graduate School of Frontier Sciences, Department of Computational Biology and Medical Sciences, and
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- Toshiki Watanabe
- Graduate School of Frontier Sciences, Department of Computational Biology and Medical Sciences, and
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説明
<jats:title>Key Points</jats:title><jats:p>ATL involves genome-wide reprogramming of the H3K27me3 pattern that is distinct from other cell types. Druggable epigenetic mechanisms are associated with ATL cell development and HTLV-1–mediated transformation.</jats:p>
収録刊行物
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- Blood
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Blood 127 (14), 1790-1802, 2016-04-07
American Society of Hematology
