Social isolation impairs remyelination in mice through modulation of IL‐6

  • Manabu Makinodan
    Department of PsychiatryNara Medical University School of Medicine Nara Japan
  • Daisuke Ikawa
    Department of PsychiatryNara Medical University School of Medicine Nara Japan
  • Yuki Miyamoto
    Department of PharmacologyNational Research Institute for Child Health and Development Tokyo Japan
  • Junji Yamauchi
    Department of PharmacologyNational Research Institute for Child Health and Development Tokyo Japan
  • Kazuhiko Yamamuro
    Department of PsychiatryNara Medical University School of Medicine Nara Japan
  • Yasunori Yamashita
    Department of PsychiatryNara Medical University School of Medicine Nara Japan
  • Michihiro Toritsuka
    Department of PsychiatryNara Medical University School of Medicine Nara Japan
  • Sohei Kimoto
    Department of PsychiatryNara Medical University School of Medicine Nara Japan
  • Kazuki Okumura
    Department of PsychiatryNara Medical University School of Medicine Nara Japan
  • Takahira Yamauchi
    Department of PsychiatryNara Medical University School of Medicine Nara Japan
  • Shin‐ichi Fukami
    Department of PsychiatryNara Medical University School of Medicine Nara Japan
  • Hiroki Yoshino
    Department of PsychiatryNara Medical University School of Medicine Nara Japan
  • Akio Wanaka
    Department of Anatomy and NeuroscienceNara Medical University School of Medicine Nara Japan
  • Toshifumi Kishimoto
    Department of PsychiatryNara Medical University School of Medicine Nara Japan

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説明

Recent studies have revealed that social experience affects myelination. These findings have important implications for disorders that feature abnormal myelination, such as multiple sclerosis (MS), as previous studies have shown that psychosocial stress exacerbates the pathobiology of MS. However, most studies have focused on psychosocial stress during the demyelination phase of MS and have not investigated the effects of social experience on remyelination. Thus, the current study sought to determine whether social experience can alter remyelination after myelin depletion. Myelin in the mouse medial prefrontal cortex was depleted with cuprizone, and the effects of subsequent social isolation on remyelination were evaluated. Remyelination was severely impaired in socially isolated mice. Social isolation also increased IL-6 levels in the medial prefrontal cortex, and administration of an IL-6 inhibitor (ND

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