Deep sequencing-based microRNA expression signatures in head and neck squamous cell carcinoma: dual strands of pre-<i>miR</i>-150 as antitumor miRNAs

  • Keiichi Koshizuka
    Department of Functional Genomics, Chiba University Graduate School of Medicine, Chuo-Ku, Chiba, Japan
  • Nijiro Nohata
    Moores Cancer Center, University of California, San Diego, La Jolla, CA, USA
  • Toyoyuki Hanazawa
    Department of Otorhinolaryngology/Head and Neck Surgery, Chiba University Graduate School of Medicine, Chiba, Japan
  • Naoko Kikkawa
    Department of Otorhinolaryngology/Head and Neck Surgery, Chiba University Graduate School of Medicine, Chiba, Japan
  • Takayuki Arai
    Department of Functional Genomics, Chiba University Graduate School of Medicine, Chuo-Ku, Chiba, Japan
  • Atsushi Okato
    Department of Functional Genomics, Chiba University Graduate School of Medicine, Chuo-Ku, Chiba, Japan
  • Ichiro Fukumoto
    Department of Functional Genomics, Chiba University Graduate School of Medicine, Chuo-Ku, Chiba, Japan
  • Koji Katada
    Department of Otorhinolaryngology/Head and Neck Surgery, Chiba University Graduate School of Medicine, Chiba, Japan
  • Yoshitaka Okamoto
    Department of Otorhinolaryngology/Head and Neck Surgery, Chiba University Graduate School of Medicine, Chiba, Japan
  • Naohiko Seki
    Department of Functional Genomics, Chiba University Graduate School of Medicine, Chuo-Ku, Chiba, Japan

書誌事項

公開日
2017-03-17
資源種別
journal article
DOI
  • 10.18632/oncotarget.16327
公開者
Impact Journals, LLC

説明

We adopted into RNA-sequencing technologies to construct the microRNA (miRNA) expression signature of head and neck squamous cell carcinoma (HNSCC). Our signature revealed that a total of 160 miRNAs (44 upregulated and 116 downregulated) were aberrantly expressed in cancer tissues. Expression of miR-150-5p (guide strand miRNA) and miR-150-3p (passenger strand miRNA) were significantly silenced in cancer tissues, suggesting both miRNAs act as antitumor miRNAs in HNSCC cells. Ectopic expression of mature miRNAs, miR-150-5p and miR-150-3p inhibited cancer cell aggressiveness. Low expression of miR-150-5p and miR-150-3p predicted significantly shorter overall survival in patients with HNSCC (P = 0.0091 and P = 0.0386) by Kaplan-Meier survival curves analyses. We identified that integrin α3 (ITGA3), integrin α6 (ITGA6), and tenascin C (TNC) were coordinately regulated by these miRNAs in HNSCC cells. Knockdown assays using siRNAs showed that ITGA3, ITGA6 and TNC acted as cancer promoting genes in HNSCC cells. Moreover, ITGA3, ITGA6, and TNC alterations were associated with significantly poorer overall survival (P = 0.0177, P = 0.0237, and P = 0.026, respectively). Dual strands of pre-150 (miR-150-5p and miR-150-3p) functioned as antitumor miRNAs based on the miRNA expression signature of HNSCC. Identification of antitumor miR-150-mediated RNA networks may provide novel insights into pathogenesis of HNSCC.

収録刊行物

  • Oncotarget

    Oncotarget 8 (18), 30288-30304, 2017-03-17

    Impact Journals, LLC

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