Treatment response and neurofilament light chain levels with long-term patisiran in hereditary transthyretin-mediated amyloidosis with polyneuropathy: 24-month results of an open-label extension study

  • Simina Ticau
    Alnylam Pharmaceuticals, Cambridge, MA, USA
  • Emre Aldinc
    Alnylam Pharmaceuticals, Cambridge, MA, USA
  • Michael Polydefkis
    Department of Neurology, Johns Hopkins University School of Medicine, Baltimore, MD, USA
  • David Adams
    Neurology Department, AP-HP, CHU Bicêtre, Université Paris-Saclay, Paris-Saclay, France
  • Teresa Coelho
    Centro Hospitalar Universitário Santo António, European Reference Network – EUroNMD, Porto, Portugal
  • Mitsuharu Ueda
    Graduate School of Medical Sciences, Kumamoto University, Kumamoto, Japan
  • Cecilia Hale
    Alnylam Pharmaceuticals, Cambridge, MA, USA
  • John Vest
    Alnylam Pharmaceuticals, Cambridge, MA, USA
  • Paul Nioi
    Alnylam Pharmaceuticals, Cambridge, MA, USA

書誌事項

公開日
2023-07-20
資源種別
journal article
権利情報
  • http://creativecommons.org/licenses/by/4.0/
DOI
  • 10.1080/13506129.2023.2232520
  • 10.6084/m9.figshare.23715676.v1
  • 10.6084/m9.figshare.23715676
公開者
Informa UK Limited

この論文をさがす

説明

Longitudinal changes in neurofilament light chain (NfL) levels were evaluated alongside prespecified clinical assessments 24 months into the patisiran Global open-label extension (OLE) study in patients with ATTRv amyloidosis with polyneuropathy. All patients enrolled in the Global OLE, from phase III APOLLO and phase II OLE parent studies, received patisiran. Assessments included measures of polyneuropathy (modified Neuropathy Impairment Score+7 (mNIS+7)), quality of life (QOL; Norfolk QOL-Diabetic Neuropathy questionnaire (Norfolk QOL-DN)), and plasma NfL. Patients receiving patisiran in the parent study (APOLLO-patisiran, n = 137; phase II OLE-patisiran, n = 25) demonstrated sustained improvements in mNIS+7 (mean change from parent study baseline (95% confidence interval): APOLLO-patisiran −4.8 (−8.9, −0.6); phase II OLE-patisiran −5.8 (−10.5, −1.2)) and Norfolk QOL-DN (APOLLO-patisiran −2.4 (−7.2, 2.3)), and maintained reduced NfL levels at Global OLE 24 months. After initiating patisiran in the Global OLE, APOLLO-placebo patients (n = 49) demonstrated stabilized mNIS+7, improved Norfolk QOL-DN, and significantly reduced NfL levels. Patisiran continued to demonstrate an acceptable safety profile. Earlier patisiran initiation was associated with a lower exposure-adjusted mortality rate. Long-term patisiran treatment led to sustained improvements in neuropathy and QOL, with NfL demonstrating potential as a biomarker for disease progression and treatment response in ATTRv amyloidosis with polyneuropathy.

収録刊行物

  • Amyloid

    Amyloid 31 (1), 1-11, 2023-07-20

    Informa UK Limited

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