Inactivation of glutathione peroxidase by nitric oxide leads to the accumulation of H<sub>2</sub>O<sub>2</sub>and the induction of HB‐EGF via c‐Jun NH<sub>2</sub>‐terminal kinase in rat aortic smooth muscle cells

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  • Young Ho Koh
    Department of BiochemistryOsaka University Medical School2–2 YamadaokaSuitaOsaka565–0871Japan
  • Keiichiro Suzuki
    Department of BiochemistryHyogo College of MedicineNishinomiyaHyogoJapan
  • Wenyi Che
    Department of BiochemistryOsaka University Medical School2–2 YamadaokaSuitaOsaka565–0871Japan
  • Yong Seek Park
    Department of BiochemistryOsaka University Medical School2–2 YamadaokaSuitaOsaka565–0871Japan
  • Yasuhide Miyamoto
    Department of BiochemistryOsaka University Medical School2–2 YamadaokaSuitaOsaka565–0871Japan
  • Shigeki Higashiyama
    Department of BiochemistryOsaka University Medical School2–2 YamadaokaSuitaOsaka565–0871Japan
  • Naoyuki Taniguchi
    Department of BiochemistryOsaka University Medical School2–2 YamadaokaSuitaOsaka565–0871Japan

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SPECIFIC AIMSThis study addresses the hypothesis that nitric oxide (NO) up-regulates the heparin binding epidermal growth factor (EGF)-like growth factor (HB-EGF) through the accumulation of peroxi...

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