Pituitary Hyposecretion and Hypersecretion Produced by a Rathke's Cleft Cyst Presenting as a Noncystic Hypothalamic Mass

DOI PDF 2 Citations
  • Marc Wenzel
    Departments of Medicine, University of Maryland School of Medicine and Hospital, Baltimore, Maryland
  • Michael Salcman
    Departments of Neurosurgery, University of Maryland School of Medicine and Hospital, Baltimore, Maryland
  • Donald A. Kristt
    Departments of Pathology, University of Maryland School of Medicine and Hospital, Baltimore, Maryland
  • Fouad E. Gellad
    Departments of Radiology, University of Maryland School of Medicine and Hospital, Baltimore, Maryland
  • Leonard P. Kapcala
    Departments of Medicine, University of Maryland School of Medicine and Hospital, Baltimore, Maryland

Description

<jats:title>Abstract</jats:title> <jats:p>A 53-year-old man presented with polydipsia, polyuria, lightheadedness on standing, and syncope. Visual field deficits suggesting left optic tract compression and pituitary dysfunction consisting of diabetes insipidus, hyperprolactinemia, and anterior pituitary insufficiency were diagnosed. On the computed tomography scan, an isodense, ring-enhancing, parasellar mass was localized primarily in the suprasellar region and also extended into the sella. It was believed that the pituitary hormone hypersecretion and hyposecretion were due to hypothalamic dysfunction from the mass which was initially believed to be a craniopharyngioma. At surgery a Rathke's cleft cyst was resected. The unusual presentation of this Rathke's cleft cyst presenting as a hypothalamic lesion is discussed.</jats:p>

Journal

  • Neurosurgery

    Neurosurgery 24 (3), 424-428, 1989-03-01

    Ovid Technologies (Wolters Kluwer Health)

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