Microbial programming of health and disease starts during fetal life

  • Petya T. Koleva
    Department of Pediatrics University of Alberta Edmonton Alberta Canada
  • Ji‐Sun Kim
    Dalla Lana School of Public Health University of Toronto Toronto Ontario Canada
  • James A. Scott
    Dalla Lana School of Public Health University of Toronto Toronto Ontario Canada
  • Anita L. Kozyrskyj
    Department of Pediatrics University of Alberta Edmonton Alberta Canada

書誌事項

公開日
2015-12
権利情報
  • http://onlinelibrary.wiley.com/termsAndConditions#vor
DOI
  • 10.1002/bdrc.21117
公開者
Wiley

この論文をさがす

説明

<jats:p>The pioneer microbiota of the neonatal gut are essential for gut maturation, and metabolic and immunologic programming. Recent research has shown that early bacterial colonization may impact the occurrence of disease later in life (microbial programming). Despite early conflicting evidence, it has long been considered that the womb is a sterile environment and human microbial colonization begins at birth. In the last few years, several findings have reiterated the presence of microbes in infant first stool (meconium) and pointed to the existence of in utero microbial colonization of the infant gut. The dominant bacterial taxa detected in meconium specimens belong to the <jats:italic>Enterobacteriaceae</jats:italic> family (<jats:italic>Escherichia</jats:italic> genus) and lactic acid bacteria (notably members of the genera <jats:italic>Leuconostoc</jats:italic>, <jats:italic>Enterococcus</jats:italic>, and <jats:italic>Lactococcus</jats:italic>). Maternal atopy promotes dominance of <jats:italic>Enterobacteriaceae</jats:italic> in newborn meconium, which in turn may lead to respiratory problems in the infant. This microbial interaction with the host immune system may in fact, originate during fetal life. Our review evaluates the evidence for an intrauterine origin of meconium microbiota, their composition and influences, and potential clinical implications on infant health. Birth Defects Research (Part C) 105:265–277, 2015. © 2015 Wiley Periodicals, Inc.</jats:p>

収録刊行物

被引用文献 (2)*注記

もっと見る

問題の指摘

ページトップへ