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- Yuhua Zhu
- Laboratory Animal Center, Xuzhou Medical University, Xuzhou 221004, Jiangsu, China
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- Xuemei Xian
- Laboratory Animal Center, Xuzhou Medical University, Xuzhou 221004, Jiangsu, China
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- Zhenzhen Wang
- Laboratory Animal Center, Xuzhou Medical University, Xuzhou 221004, Jiangsu, China
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- Yingchao Bi
- Laboratory Animal Center, Xuzhou Medical University, Xuzhou 221004, Jiangsu, China
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- Quangang Chen
- Laboratory Animal Center, Xuzhou Medical University, Xuzhou 221004, Jiangsu, China
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- Xufeng Han
- Laboratory Animal Center, Xuzhou Medical University, Xuzhou 221004, Jiangsu, China
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- Daoquan Tang
- Laboratory Animal Center, Xuzhou Medical University, Xuzhou 221004, Jiangsu, China
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- Renjin Chen
- Laboratory Animal Center, Xuzhou Medical University, Xuzhou 221004, Jiangsu, China
説明
<jats:p>Atherosclerosis is a chronic inflammatory disease; unstable atherosclerotic plaque rupture, vascular stenosis, or occlusion caused by platelet aggregation and thrombosis lead to acute cardiovascular disease. Atherosclerosis-related inflammation is mediated by proinflammatory cytokines, inflammatory signaling pathways, bioactive lipids, and adhesion molecules. This review discusses the effects of inflammation and the systemic inflammatory signaling pathway on atherosclerosis, the role of related signaling pathways in inflammation, the formation of atherosclerosis plaques, and the prospects of treating atherosclerosis by inhibiting inflammation.</jats:p>
収録刊行物
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- Biomolecules
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Biomolecules 8 (3), 80-, 2018-08-23
MDPI AG
