Role of Reactive Oxygen Species in the Progression of Type 2 Diabetes and Atherosclerosis

  • Hideaki Kaneto
    Department of Metabolic Medicine, Graduate School of Medicine, Osaka University, 2-2 Yamadaoka, Suita, Osaka 565-0871, Japan
  • Naoto Katakami
    Department of Metabolic Medicine, Graduate School of Medicine, Osaka University, 2-2 Yamadaoka, Suita, Osaka 565-0871, Japan
  • Munehide Matsuhisa
    Department of Metabolic Medicine, Graduate School of Medicine, Osaka University, 2-2 Yamadaoka, Suita, Osaka 565-0871, Japan
  • Taka-aki Matsuoka
    Department of Metabolic Medicine, Graduate School of Medicine, Osaka University, 2-2 Yamadaoka, Suita, Osaka 565-0871, Japan

書誌事項

公開日
2010
権利情報
  • http://creativecommons.org/licenses/by/3.0/
DOI
  • 10.1155/2010/453892
公開者
Wiley

この論文をさがす

説明

<jats:p>Type 2 diabetes is the most prevalent and serious metabolic disease all over the world, and its hallmarks are pancreatic -cell dysfunction and insulin resistance. Under diabetic conditions, chronic hyperglycemia and subsequent augmentation of reactive oxygen species (ROS) deteriorate -cell function and increase insulin resistance which leads to the aggravation of type 2 diabetes. In addition, chronic hyperglycemia and ROS are also involved in the development of atherosclerosis which is often observed under diabetic conditions. Taken together, it is likely that ROS play an important role in the development of type 2 diabetes and atherosclerosis.</jats:p>

収録刊行物

被引用文献 (9)*注記

もっと見る

詳細情報 詳細情報について

問題の指摘

ページトップへ