Protein kinase A–dependent phosphorylation stimulates the transcriptional activity of hypoxia-inducible factor 1
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- John W. Bullen
- Vascular Program, Institute for Cell Engineering, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA.
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- Irina Tchernyshyov
- Department of Medicine, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA.
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- Ronald J. Holewinski
- Department of Medicine, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA.
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- Lauren DeVine
- Department of Biological Chemistry, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA.
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- Fan Wu
- Department of Medicine, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA.
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- Vidya Venkatraman
- Department of Medicine, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA.
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- David L. Kass
- Department of Medicine, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA.
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- Robert N. Cole
- Department of Biological Chemistry, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA.
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- Jennifer Van Eyk
- Department of Medicine, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA.
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- Gregg L. Semenza
- Vascular Program, Institute for Cell Engineering, Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA.
抄録
<jats:p>Activation of PKA in cancer cells or cardiomyocytes results in the increased expression of HIF-1 target genes.</jats:p>
収録刊行物
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- Science Signaling
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Science Signaling 9 (430), ra56-, 2016-05-31
American Association for the Advancement of Science (AAAS)