Cutting Edge: Regulatory T Cells from Lung Cancer Patients Directly Inhibit Autologous T Cell Proliferation

  • Edward Y Woo
    Department of Surgery, University of Pennsylvania Medical Center , Philadelphia, PA 19104
  • Heidi Yeh
    Department of Surgery, University of Pennsylvania Medical Center , Philadelphia, PA 19104
  • Christina S Chu
    Department of Obstetrics and Gynecology, University of Pennsylvania Medical Center , Philadelphia, PA 19104
  • Katia Schlienger
    Abramson Family Cancer Research Institute, University of Pennsylvania Medical Center , Philadelphia, PA 19104
  • Richard G Carroll
    Abramson Family Cancer Research Institute, University of Pennsylvania Medical Center , Philadelphia, PA 19104
  • James L Riley
    Abramson Family Cancer Research Institute, University of Pennsylvania Medical Center , Philadelphia, PA 19104
  • Larry R Kaiser
    Department of Surgery, University of Pennsylvania Medical Center , Philadelphia, PA 19104
  • Carl H June
    Abramson Family Cancer Research Institute, University of Pennsylvania Medical Center , Philadelphia, PA 19104

書誌事項

公開日
2002-05-01
権利情報
  • https://academic.oup.com/pages/standard-publication-reuse-rights
DOI
  • 10.4049/jimmunol.168.9.4272
公開者
Oxford University Press (OUP)

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説明

<jats:title>Abstract</jats:title> <jats:p>Active suppression by T regulatory cells plays an important role in the down-regulation of T cell responses to foreign and self-Ags. Thus far, the potential role of CD4+CD25+ T cells in human tumors has not been reported. In this work we show that lung tumors contain large numbers of these cells and that they have constitutive high-level expression of CD152 (CTLA-4). Furthermore, the CD4+CD25+ T cells mediate potent inhibition of autologous T cell proliferation. Finally, regulatory T cells from patient tumors failed to inhibit the proliferation of allogeneic T cells. Together these results suggest that the CD4+CD25+ T cells found in lung tumors selectively inhibit the host immune response and therefore could contribute to the progression of lung cancer.</jats:p>

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