PCP and Septins Compartmentalize Cortical Actomyosin to Direct Collective Cell Movement

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  • Asako Shindo
    Howard Hughes Medical Institute and University of Texas at Austin, Austin, TX 78712, USA.
  • John B. Wallingford
    Howard Hughes Medical Institute and University of Texas at Austin, Austin, TX 78712, USA.

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<jats:title>Sculpting Actomyosin</jats:title> <jats:p> The sculpting of embryos during development involves coordinated movement of cells in large groups. How actomyosin is controlled during such collective cell movement remains poorly understood. Working with developing <jats:italic>Xenopus</jats:italic> mesoderm, <jats:bold>Shindo and Wallingford</jats:bold> (p. <jats:related-article xmlns:xlink="http://www.w3.org/1999/xlink" ext-link-type="doi" issue="6171" page="649" related-article-type="in-this-issue" vol="343" xlink:href="10.1126/science.1243126">649</jats:related-article> ) found that planar cell polarity proteins and septins interface with the actomyosin machinery to control collective cell movement. </jats:p>

Journal

  • Science

    Science 343 (6171), 649-652, 2014-02-07

    American Association for the Advancement of Science (AAAS)

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