Intercellular calcium signaling mediated by point-source burst release of ATP

  • Gregory Arcuino
    Department of Cell Biology, Anatomy, and Pathology, New York Medical College, Valhalla, NY 10595
  • Jane H.-C. Lin
    Department of Cell Biology, Anatomy, and Pathology, New York Medical College, Valhalla, NY 10595
  • Takahiro Takano
    Department of Cell Biology, Anatomy, and Pathology, New York Medical College, Valhalla, NY 10595
  • Collins Liu
    Department of Cell Biology, Anatomy, and Pathology, New York Medical College, Valhalla, NY 10595
  • Li Jiang
    Department of Cell Biology, Anatomy, and Pathology, New York Medical College, Valhalla, NY 10595
  • Qun Gao
    Department of Cell Biology, Anatomy, and Pathology, New York Medical College, Valhalla, NY 10595
  • Jian Kang
    Department of Cell Biology, Anatomy, and Pathology, New York Medical College, Valhalla, NY 10595
  • Maiken Nedergaard
    Department of Cell Biology, Anatomy, and Pathology, New York Medical College, Valhalla, NY 10595

書誌事項

公開日
2002-07-03
DOI
  • 10.1073/pnas.152588599
公開者
Proceedings of the National Academy of Sciences

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説明

<jats:p>Calcium signaling, manifested as intercellular waves of rising cytosolic calcium, is, in many cell types, the result of calcium-induced secretion of ATP and activation of purinergic receptors. The mechanism by which ATP is released has hitherto not been established. Here, we show by real-time bioluminescence imaging that ATP efflux is not uniform across a field of cells but is restricted to brief, abrupt point-source bursts. The ATP bursts emanate from single cells and manifest the transient opening of nonselective membrane channels, which admits fluorescent indicators of ≤1.5 kDa. These observations challenge the existence of regenerative ATP release, because ATP efflux is finite and restricted to a point source. Transient efflux of cytosolic nucleotides from a subset of cells may represent a conserved pathway for coordinating local activity of electrically nonexcitable cells, because identical patterns of ATP release were identified in human astrocytes, endothelial cells, and bronchial epithelial cells.</jats:p>

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