Kinase Domain Activation of FGFR2 Yields High-Grade Lung Adenocarcinoma Sensitive to a Pan-FGFR Inhibitor in a Mouse Model of NSCLC

  • Jeremy H. Tchaicha
    1Department of Medicine, Dana Farber Cancer Institute, Boston, Massachusetts.
  • Esra A. Akbay
    1Department of Medicine, Dana Farber Cancer Institute, Boston, Massachusetts.
  • Abigail Altabef
    1Department of Medicine, Dana Farber Cancer Institute, Boston, Massachusetts.
  • Oliver R. Mikse
    1Department of Medicine, Dana Farber Cancer Institute, Boston, Massachusetts.
  • Eiki Kikuchi
    1Department of Medicine, Dana Farber Cancer Institute, Boston, Massachusetts.
  • Kevin Rhee
    1Department of Medicine, Dana Farber Cancer Institute, Boston, Massachusetts.
  • Rachel G. Liao
    1Department of Medicine, Dana Farber Cancer Institute, Boston, Massachusetts.
  • Roderick T. Bronson
    5Department of Microbiology and Immunobiology, Division of Immunology, Harvard Medical School, Boston, Massachusetts.
  • Lynette M. Sholl
    6Department of Pathology, Brigham and Women's Hospital, Boston, Massachusetts.
  • Matthew Meyerson
    1Department of Medicine, Dana Farber Cancer Institute, Boston, Massachusetts.
  • Peter S. Hammerman
    1Department of Medicine, Dana Farber Cancer Institute, Boston, Massachusetts.
  • Kwok-Kin Wong
    1Department of Medicine, Dana Farber Cancer Institute, Boston, Massachusetts.

説明

<jats:title>Abstract</jats:title> <jats:p>Somatic mutations in FGFR2 are present in 4% to 5% of patients diagnosed with non–small cell lung cancer (NSCLC). Amplification and mutations in FGFR genes have been identified in patients with NSCLCs, and clinical trials are testing the efficacy of anti-FGFR therapies. FGFR2 and other FGFR kinase family gene alterations have been found in both lung squamous cell carcinoma and lung adenocarcinoma, although mouse models of FGFR-driven lung cancers have not been reported. Here, we generated a genetically engineered mouse model (GEMM) of NSCLC driven by a kinase domain mutation in FGFR2. Combined with p53 ablation, primary grade 3/4 adenocarcinoma was induced in the lung epithelial compartment exhibiting locally invasive and pleiotropic tendencies largely made up of multinucleated cells. Tumors were acutely sensitive to pan-FGFR inhibition. This is the first FGFR2-driven lung cancer GEMM, which can be applied across different cancer indications in a preclinical setting. Cancer Res; 74(17); 4676–84. ©2014 AACR.</jats:p>

収録刊行物

  • Cancer Research

    Cancer Research 74 (17), 4676-4684, 2014-09-01

    American Association for Cancer Research (AACR)

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