Genomic Analyses of Acute Flaccid Myelitis Cases among a Cluster in Arizona Provide Further Evidence of Enterovirus D68 Role

  • Jolene R. Bowers
    Pathogen and Microbiome Division, Translational Genomics Research Institute, Flagstaff, Arizona, USA
  • Michael Valentine
    Pathogen and Microbiome Division, Translational Genomics Research Institute, Flagstaff, Arizona, USA
  • Veronica Harrison
    Pathogen and Microbiome Division, Translational Genomics Research Institute, Flagstaff, Arizona, USA
  • Viacheslav Y. Fofanov
    School of Informatics Computing and Cyber Systems, Northern Arizona University, Flagstaff, Arizona, USA
  • John Gillece
    Pathogen and Microbiome Division, Translational Genomics Research Institute, Flagstaff, Arizona, USA
  • Josie Delisle
    Pathogen and Microbiome Division, Translational Genomics Research Institute, Flagstaff, Arizona, USA
  • Bethany Patton
    Pathogen and Microbiome Division, Translational Genomics Research Institute, Flagstaff, Arizona, USA
  • James Schupp
    Pathogen and Microbiome Division, Translational Genomics Research Institute, Flagstaff, Arizona, USA
  • Krystal Sheridan
    Pathogen and Microbiome Division, Translational Genomics Research Institute, Flagstaff, Arizona, USA
  • Darrin Lemmer
    Pathogen and Microbiome Division, Translational Genomics Research Institute, Flagstaff, Arizona, USA
  • Scott Ostdiek
    Phoenix Children’s Hospital, Phoenix, Arizona, USA
  • Harlori K. Bains
    Phoenix Children’s Hospital, Phoenix, Arizona, USA
  • Jennifer Heim
    Phoenix Children’s Hospital, Phoenix, Arizona, USA
  • Tammy Sylvester
    Maricopa County Department of Public Health, Phoenix, Arizona, USA
  • Siru Prasai
    Maricopa County Department of Public Health, Phoenix, Arizona, USA
  • Melissa Kretschmer
    Maricopa County Department of Public Health, Phoenix, Arizona, USA
  • Nicole Fowle
    Maricopa County Department of Public Health, Phoenix, Arizona, USA
  • Kenneth Komatsu
    Arizona Department of Health Services, Phoenix, Arizona, USA
  • Shane Brady
    Arizona Department of Health Services, Phoenix, Arizona, USA
  • Susan Robinson
    Arizona Department of Health Services, Phoenix, Arizona, USA
  • Kathryn Fitzpatrick
    Arizona Department of Health Services, Phoenix, Arizona, USA
  • Gholamabbas Amin Ostovar
    Maricopa Integrated Health System, Phoenix, Arizona, USA
  • Eric Alsop
    Neurogenomics Division, Translational Genomics Research Institute, Phoenix, Arizona, USA
  • Elizabeth Hutchins
    Neurogenomics Division, Translational Genomics Research Institute, Phoenix, Arizona, USA
  • Kendall Jensen
    Neurogenomics Division, Translational Genomics Research Institute, Phoenix, Arizona, USA
  • Paul Keim
    Pathogen and Microbiome Division, Translational Genomics Research Institute, Flagstaff, Arizona, USA
  • David M. Engelthaler
    Pathogen and Microbiome Division, Translational Genomics Research Institute, Flagstaff, Arizona, USA

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説明

<jats:p>Enteroviruses frequently result in respiratory and gastrointestinal illness; however, multiple subtypes, including poliovirus, can cause severe neurologic disease. Recent biennial increases (i.e., 2014, 2016, and 2018) in cases of non-polio acute flaccid paralysis have led to speculations that other enteroviruses, specifically enterovirus D68 (EV-D68), are emerging to fill the niche that was left from poliovirus eradication. A cluster of 11 suspect cases of pediatric acute flaccid myelitis (AFM) was identified in 2016 in Phoenix, AZ. Multiple genomic analyses identified the presence of EV-D68 in the majority of clinical AFM cases. Beyond limited detection of herpesvirus, no other likely etiologies were found in the cluster. These findings strengthen the likelihood that EV-D68 is a cause of AFM and show that the rapid molecular assays developed for this study are useful for investigations of AFM and EV-D68.</jats:p>

収録刊行物

  • mBio

    mBio 10 (1), e02262-, 2019-02-26

    American Society for Microbiology

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