Biological activities of deoxyspergualin in autoimmune disease mice.

  • NEMOTO KYUICHI
    Research Laboratories, Pharmaceuticals Group, Nippon Kayaku Co., Ltd.
  • HAYASHI MICHIKO
    Research Laboratories, Pharmaceuticals Group, Nippon Kayaku Co., Ltd.
  • SUGAWARA YUMI
    Research Laboratories, Pharmaceuticals Group, Nippon Kayaku Co., Ltd.
  • ITO JUNPEI
    Research Laboratories, Pharmaceuticals Group, Nippon Kayaku Co., Ltd.
  • ABE FUMINORI
    Research Laboratories, Pharmaceuticals Group, Nippon Kayaku Co., Ltd.
  • TAKITA TOMOHISA
    Research Laboratories, Pharmaceuticals Group, Nippon Kayaku Co., Ltd.
  • NAKAMURA TERUYA
    Central Research Laboratories, Takara Shuzo Co., Ltd.
  • TAKEUCHI TOMIO
    Institute of Microbial Chemistry

この論文をさがす

説明

An assessment of the prophylactic and ameliorative effects of deoxyspergualin (NKT-01), an immunosuppressive agent, was carried out in male MRL/MpJ-1pr/1pr (MRL/1) mice which spontaneously develop lupus-like lesions. When NKT-01 was administered ip daily from the age of either 8 or 19 weeks, diseases such as massive lymphadenopathy, circulating anti-DNA antibody and lupus nephritis were markedly suppressed. The primary response to lipopolysaccharide was significantly reduced in MRL/1 mice administered NKT-01 but the response to sheep red blood cells was not affected. The ability of spleen cells to release interleukins 2 and 3 with or without mitogen was significantly enhanced in mice receiving NKT-01. These findings demonstrate that NKT-01 has therapeutic activity against the development of spontaneous disease in MRL/1 mice.

収録刊行物

キーワード

詳細情報 詳細情報について

  • CRID
    1390001204151206656
  • NII論文ID
    130003501492
  • DOI
    10.7164/antibiotics.41.1253
  • COI
    1:CAS:528:DyaL1cXlvVehs7w%3D
  • ISSN
    18811469
    00218820
  • PubMed
    3263348
  • 本文言語コード
    en
  • 資料種別
    journal article
  • データソース種別
    • JaLC
    • Crossref
    • PubMed
    • CiNii Articles
  • 抄録ライセンスフラグ
    使用不可

問題の指摘

ページトップへ