脳性麻痺の原因

DOI

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タイトル別名
  • Etiology of Cerebral Palsy

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Two years follow up study of sixty-two cases of neonatal cerebral damage are reported.<BR>Nineteen of the thirty-six survived cases in series had cerebral palsy.<BR>These cases include, seven with hyperbilirubinemia, five with asphyxia, three with intracranial hemorrhage, one with intracranial hemorrhage and asphyxia, one with intracranial hemorrhage with hypoglycemia and one with hypoglycemia and hypocalcemia.<BR>The causes of hyper bilirubinemia include ABO incompatibility, administration of Salcylate and idiopathic origin.<BR>Kernicterus occurred in cases with bilirubin level over 20mg/d/ in full term infants, whereas 14 mg/dl in premature infants with asphyxia or hypoglycemia.<BR>Fifty per cent of anoxic encephalopathies in the neonatal period had cerebral palsy. In group whose convulsive seizure improved within one day, there had no cerebral palsy, whereas in group whose seizure last more than 4 days, all had cerebral palsy.<BR>All cases of intracranial hemorrhage and two of the three cases of neonatal hypoglycemia had cerebral palsy.<BR>The autopsy findings of one case of cerebral palsy caused by neonatal asphyxia revealed the nerve cell destruction and the gliosis of the basal ganglions but there was no pathological finding in the cerebral cortex.

収録刊行物

  • 脳と発達

    脳と発達 3 (4), 333-343, 1971

    THE JAPANESE SOCIETY OF CHILD NEUROLOGY

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