A case of recurrent stroke associated with chronic Stanford type B aortic dissection

  • Tonomura Shuichi
    Department of Neurology, National Cerebral and Caridovascular Center
  • Saito Kozue
    Department of Neurology, National Cerebral and Caridovascular Center
  • Tanaka Tomotaka
    Department of Neurology, National Cerebral and Caridovascular Center
  • Nishimura Takuya
    Department of Neurology, National Cerebral and Caridovascular Center
  • Kinoshita Naoto
    Department of Neurology, National Cerebral and Caridovascular Center
  • Higashida Kyoko
    Department of Neurology, National Cerebral and Caridovascular Center
  • Fukuma Kazuki
    Department of Neurology, National Cerebral and Caridovascular Center
  • Okuno Yoshinori
    Department of Neurology, National Cerebral and Caridovascular Center
  • Sugiura Yuri
    Department of Neurology, National Cerebral and Caridovascular Center
  • Takasugi Junji
    Department of Neurology, National Cerebral and Caridovascular Center
  • Motoyama Rie
    Department of Neurology, National Cerebral and Caridovascular Center
  • Yamagami Hiroshi
    Department of Neurology, National Cerebral and Caridovascular Center
  • Nagatsuka Kazuyuki
    Department of Neurology, National Cerebral and Caridovascular Center

Bibliographic Information

Other Title
  • スタンフォードB 型大動脈解離慢性期に左椎骨動脈領域に脳梗塞を繰り返した1 例

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Description

A 67-year-old man was admitted to our hospital with a sudden onset of left dominant quadriparesis and left sensory impairment. The patient had diagnosed with Stanford type B aortic dissection (AD) and ischemic stroke in the left posterior inferior cerebellar artery (PICA) territory 1 year before his admission. Magnetic resonance imaging revealed acute ischemic stroke in the left PICA and anterior spinal artery territory. Transesophageal echography (TEE) was performed to identify the etiology. The false lumen of the type B AD was partially thrombosed, and the primary intimal tear was found to be close to the origin of the left vertebral artery (VA), which arose from proximal portion of left subclavian artery. Moreover, color Doppler TEE revealed a retrograde flow from the false lumen to the left VA. We considered that the thrombus in the false lumen of the type B AD could be an embolic source of repetitive strokes of the left VA territory. The patient was treated with anticoagulant therapy to prevent the formation of thrombus in the false lumen. No recurrence was observed until 1 year of follow-up.

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