Maize plants mutated in NAD(P)H-dependent HC-toxin reductase gene (Hm1) is vulnerable to H2O2 stress
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- Hayashi Mitsunori
- Institute of Molecular and Cellular Biosciences, The University of Tokyo
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- Takahashi Hideyuki
- Institute of Molecular and Cellular Biosciences, The University of Tokyo
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- Kawai-Yamada Maki
- Institute of Molecular and Cellular Biosciences, The University of Tokyo
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- Tezuka Takafumi
- Biological Science, University of Nagoya
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- Uchimiya Hirofumi
- Institute of Molecular and Cellular Biosciences, The University of Tokyo
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Abstract
Maize Hm1 gene encodes a NAD(P)H-dependent HC-toxin reductase, which detoxify HC-toxin produced by fungus Cochliobolus carbonum (Meeley and Walton 1991 Plant Phys 97: 1080). Measurements of ion leakage indicated that H2O2 treatment of a recessive mutant (hm1) of maize resulted in accelerated death in excised leaves. Furthermore, an hm1 maize showed quantitative decrease of NAD(H) level. Thus, the Hm1 gene may confer other functions related to ROS stress tolerance.
Journal
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- Plant Biotechnology
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Plant Biotechnology 22 (1), 69-70, 2005
Japanese Society for Plant Biotechnology
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Details 詳細情報について
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- CRID
- 1390282679305091456
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- NII Article ID
- 10026526470
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- NII Book ID
- AA11250821
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- ISSN
- 13476114
- 13424580
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- Text Lang
- en
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- Data Source
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- JaLC
- Crossref
- CiNii Articles
- KAKEN
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- Abstract License Flag
- Disallowed