A CASE OF BRONCHIAL ASTHMA CAUSED BY OCCUPATIONAL EXPOSURE TO TRICHOPHYTON

  • Hoshi Rie
    Allergy Center, Saitama Medical University
  • Nakagome Kazuyuki
    Allergy Center, Saitama Medical University:Department of Respiratory Medicine, Saitama Medical University
  • Aoki Hirotoshi
    Allergy Center, Saitama Medical University
  • Takaku Yotaro
    Allergy Center, Saitama Medical University:Department of Respiratory Medicine, Saitama Medical University
  • Yamaguchi Takefumi
    Allergy Center, Saitama Medical University:Department of Respiratory Medicine, Saitama Medical University
  • Soma Tomoyuki
    Allergy Center, Saitama Medical University:Department of Respiratory Medicine, Saitama Medical University
  • Nishihara Fuyumi
    Allergy Center, Saitama Medical University:Department of Respiratory Medicine, Saitama Medical University
  • Komiyama Ken-ichiro
    Allergy Center, Saitama Medical University:Department of Respiratory Medicine, Saitama Medical University
  • Hagiwara Kouichi
    Department of Respiratory Medicine, Saitama Medical University
  • Kanazawa Minoru
    Department of Respiratory Medicine, Saitama Medical University
  • Nagata Makoto
    Allergy Center, Saitama Medical University:Department of Respiratory Medicine, Saitama Medical University

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Other Title
  • トリコフィトンの職業性曝露が増悪の原因と考えられた気管支喘息の1例
  • 症例報告 トリコフィトンの職業性曝露が増悪の原因と考えられた気管支喘息の1例
  • ショウレイ ホウコク トリコフィトン ノ ショクギョウセイ バクロ ガ ゾウアク ノ ゲンイン ト カンガエラレタ キカンシ ゼンソク ノ 1レイ

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A case involved a 39-year-old female nurse in a health-care facility for elderly individuals requiring long-term care, who presented with insufficient control of bronchial asthma. Although she did not have tinea, she had opportunities for contact with patients who did. Careful interview of history suggested a relationship between asthma exacerbation and workplace, so we measured the specific IgE antibody to Trichophyton and confirmed a positive result. As occupational exposure to Trichophyton was considered as a cause of asthma exacerbations, avoidance of Trichophyton as well as antiasthma treatment was conducted and symptoms improved. Identification and avoidance of specific allergens is essential for successful long-term management of asthma. However, measurement of specific IgE antibody to Trichophyton is not routinely performed, although this fungus could induce not only tinea, but also asthma. The possibility that occupational exposure to trichophyton could exacerbate asthma symptoms needs to be kept in mind, particularly in the case of nurses who may be in contact with elderly individuals with tinea.

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