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- NAKAMURA Yoshiro
- Department of Internal Medicine, School of Medicine, Keio University Nomura Medical Center of Health Care
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- TAKAHASHI Masando
- Department of Internal Medicine, School of Medicine, Keio University Nomura Medical Center of Health Care
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- HATTORI Shigehiko
- Department of Internal Medicine, School of Medicine, Keio University Nomura Medical Center of Health Care
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抄録
Effect of increased left atrial pressure and a positive inotropic agent upon asynergic left ventricle which was produced by acute occlusion of LAD were examined in 8 open-chest dogs. Systolic bulging in ischemic area was qualified by ESL/EDL in percentage. Left atrial pressure was controlled by the reservoir connected to left atrial appendage with a large bore tubing. Cardiac output and aortic pressure were decreased following coronary occlusion without change in isometric developed tension in nonischemic area of left ventricle. Increase of left atrial pressure from 5mmHg to 14mmHg produced rise in stroke volume to 191% and rise in aortic pressure to 156% in asynergic left ventricle respectively. These increases were not accompanied by change in ESL/EDL.<br>Therefore, extracardiac factors were assumed to explain the prominent increase in left ventricular stroke work by elevation of left atrial pressure in asynergic left ventricle. Isoproterenol enhanced total cardiac performance by increasing the function of nonischemic area without deterious effect on the ischemic area.
収録刊行物
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- Japanese Heart Journal
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Japanese Heart Journal 18 (5), 729-735, 1977
International Heart Journal刊行会
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詳細情報 詳細情報について
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- CRID
- 1390282680014924160
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- NII論文ID
- 130000762881
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- NII書誌ID
- AA00690786
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- ISSN
- 1348673X
- 00214868
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- NDL書誌ID
- 1912910
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- PubMed
- 926317
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- 本文言語コード
- en
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- データソース種別
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- JaLC
- NDL
- Crossref
- PubMed
- CiNii Articles
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- 抄録ライセンスフラグ
- 使用不可