A Case of Paroxysmal Kinesigenic Dyskinesia with Complex Partial Seizure Probably Associated with Amygdala Enlargement

DOI 参考文献11件 オープンアクセス
  • Mogi Taichi
    Department of Psychiatry, National Center of Neurology and Psychiatry, Tokyo, Japan
  • Komori Takashi
    Department of Laboratory Medicine and Pathology, Tokyo Metropolitan Neurological Hospital, Tokyo, Japan
  • Morino Michiharu
    Department of Neurosurgery, Tokyo Metropolitan Neurological Hospital, Tokyo, Japan
  • Sone Daichi
    Department of Psychiatry, National Center of Neurology and Psychiatry, Tokyo, Japan
  • Watanabe Masako
    Department of Psychiatry, National Center of Neurology and Psychiatry, Tokyo, Japan
  • Taniguchi Go
    Department of Psychiatry, Tokyo University, Tokyo, Japan
  • Murata Yoshiko
    Department of Psychiatry, National Center of Neurology and Psychiatry, Tokyo, Japan
  • Watanabe Yutaka
    Department of Psychiatry, National Center of Neurology and Psychiatry, Tokyo, Japan

書誌事項

公開日
2014
DOI
  • 10.3805/eands.7.1
公開者
一般社団法人 日本てんかん学会

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説明

Paroxysmal kinesigenic dyskinesia (PKD) is a rare disease characterized by involuntary movements triggered by a sudden initiation of voluntary movement. No EEG abnormalities are observed both during and between PKD attacks. Clinically, the involuntary movements are readily controlled by sodium channel-related antiepileptic drugs such as carbamazepine [1] and phenytoin [2]. Two hypotheses have been proposed for the pathophysiology of PKD; kinesthetic reflex epilepsy [3] and basal ganglia functional abnormality [4], but no consensus has been reached.<br>We report a patient with amygdala enlargement who manifested complex partial seizure (CPS) following PKD attack.

収録刊行物

  • Epilepsy & Seizure

    Epilepsy & Seizure 7 (1), 1-13, 2014

    一般社団法人 日本てんかん学会

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