Three Distinct Commonly Deleted Regions of Chromosome Arm 16q in Human Primary and Metastatic Prostate Cancers
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- Suzuki Hiroyoshi
- Department of Urology, School of Medicine, Chiba University
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- Komiya Akira
- Department of Urology, School of Medicine, Chiba University
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- Emi Mitsuru
- Department of Molecular Biology, Institute of Gerontology, Nippon Medical School
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- Kuramochi Hiroaki
- Department of Urology, School of Medicine, Chiba University
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- Shiraishi Taizo
- Department of Pathology, Faculty of Medicine, Mie University
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- Yatani Ryuichi
- Department of Pathology, Faculty of Medicine, Mie University
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- Shimazaki Jun
- Department of Urology, School of Medicine, Chiba University
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説明
Human prostate cancers frequently show loss of heterozygosity (LOH) at loci on the long arm of chromosome 16 (16q). In this study, we analyzed prostate cancer specimens from 48 patients (Stage B, 20 cases; Stage C, 10 cases; cancer death, 18 cases) for allelic loss on 16q, using either restriction fragment length polymorphism (RFLP)-or polymerase chain reaction (PCR)-based methods. Allelic losses were observed in 20 (42%) of 48 cases, all of which were informative with at least one locus. Detailed deletion mapping identified three distinct commonly deleted regions on this chromosome arm : q22.1-q22.3,q23.2-q24.1,and q24.3-qter. On the basis of a published sex-averaged framework map, the estimated sizes of the commonly deleted regions were 4.7 (16q22.1-q22.3), 17.2 (16q23.2-q24.1) and 8.4 cM (16q24.3-qter). Allelic losses on 16q were observed more frequently in the cancer-death cases (11 of 18; 61%) than in early-stage tumor cases (9 of 30; 30%; P<0.05). In 7 of 11 patients from whom DNA was available from metastatic cancers as well as from normal tissues and primary tumors, the primary cancer foci had no detectable abnormality of 16q, but the metastatic tumors showed LOH. These results suggest that inactivation of tumor suppressor genes on 16q plays an important role in the progression of prostate cancer. We also analyzed exons 5-8 of the E-cadherin gene, located at 16q22.1,in tumor DNA by means of PCR-single strand conformation polymorphism and direct sequencing, but we detected no somatic mutations in this candidate gene. Genes Chromosom Cancer 17 : 225-233 (1996).
収録刊行物
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- 日本医科大学老人病研究所紀要
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日本医科大学老人病研究所紀要 3 79-87, 1997-03-25
日本医科大学
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詳細情報 詳細情報について
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- CRID
- 1571135651874946304
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- NII論文ID
- 110001798164
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- NII書誌ID
- AN1047681X
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- ISSN
- 13409662
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- 本文言語コード
- en
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- データソース種別
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