<Originals>Pressure-Volume Curves in Pulmonary Edema

  • TSUYA Yasuo
    Fourth Department of Internal Medicine, Kinki University School of Medicine
  • OHISHI Mitsuo
    Fourth Department of Internal Medicine, Kinki University School of Medicine
  • TAKAGI Ohmi
    Fourth Department of Internal Medicine, Kinki University School of Medicine
  • UENO Hiroshi
    Fourth Department of Internal Medicine, Kinki University School of Medicine
  • FUJIHARA Yoshimasa
    Fourth Department of Internal Medicine, Kinki University School of Medicine
  • NAKAJIMA Shigenori
    Fourth Department of Internal Medicine, Kinki University School of Medicine

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  • <原著>肺水腫の肺圧量曲線について

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Pulmonary edema, pulmonary interstitial edema and hydration lung were prepared experimentally. By using P-V curves, elastic recoil of each pulmonary tissue, change of lung surface tension of each case were examined, and the mechanism of the change from pulmonary interstitial edema to pulmonary edema was studied. In pulmonary interstitial edema, the lung compliance was not reduced, and the TLC was rather increased. However, severe pulmonary edema reduced the lung compliance and also reduced the TLC due to collapse of the lung. Interstitial edema had no influence on the lung surface tension. Severe pulmonary edema increased the surface tension only at high lung volume, but had few influences on it at low lung volume. In the dehydration lung, the lung compliance was not decreased at all, but rather increased near the total lung capacity. "Air trapping" was often found in severe pulmonary edema. It was suggested that an interstitial edema around the bronchiole was playing an important role on the phenomenon.

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