Therapeutic effects of KCC2 chloride transporter activation on detrusor overactivity in mice with spinal cord injury
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- Kyohei Watanabe
- Department of Neurophysiology, Hamamatsu University School of Medicine, Hamamatsu, Japan
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- Masaru Ishibashi
- Department of Neurophysiology, Hamamatsu University School of Medicine, Hamamatsu, Japan
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- Takahisa Suzuki
- Department of Urology, Kanagawa Rehabilitation Hospital, Atsugi, Japan
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- Atsushi Otsuka
- Department of Urology, Hamamatsu University School of Medicine, Hamamatsu, Japan
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- Naoki Yoshimura
- Department of Urology, University of Pittsburgh School of Medicine, Pittsburgh, Pennsylvania, United States
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- Hideaki Miyake
- Department of Urology, Hamamatsu University School of Medicine, Hamamatsu, Japan
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- Atsuo Fukuda
- Department of Neurophysiology, Hamamatsu University School of Medicine, Hamamatsu, Japan
抄録
<jats:p> This study is the first report to suggest that activation of the Cl<jats:sup>−</jats:sup> transporter K<jats:sup>+</jats:sup>-Cl<jats:sup>−</jats:sup> cotransporter 2 may be a therapeutic modality for the treatment of spinal cord injury-induced detrusor overactivity by targeting bladder efferent pathways. </jats:p>
収録刊行物
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- American Journal of Physiology-Renal Physiology
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American Journal of Physiology-Renal Physiology 324 (4), F353-F361, 2023-04-01
American Physiological Society
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キーワード
詳細情報 詳細情報について
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- CRID
- 1360861705568147328
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- ISSN
- 15221466
- 1931857X
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- データソース種別
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- Crossref
- KAKEN