Extracellular Ca
            <sup>2+</sup>
            Acts as a Mediator of Communication from Neurons to Glia

DOI Web Site 4 Citations

Arnulfo Torres

Center for Translational Neuromedicine, Division of Glial Disease and Therapeutics, University of Rochester Medical School, 601 Elmwood Avenue, Rochester, NY 14642, USA.
Fushun Wang

Center for Translational Neuromedicine, Division of Glial Disease and Therapeutics, University of Rochester Medical School, 601 Elmwood Avenue, Rochester, NY 14642, USA.
Qiwu Xu

Center for Translational Neuromedicine, Division of Glial Disease and Therapeutics, University of Rochester Medical School, 601 Elmwood Avenue, Rochester, NY 14642, USA.
Takumi Fujita

Center for Translational Neuromedicine, Division of Glial Disease and Therapeutics, University of Rochester Medical School, 601 Elmwood Avenue, Rochester, NY 14642, USA.
Radoslaw Dobrowolski

Institut fuer Genetik, Rheinische Friedrich-Wilhelms-Universitaet, D-53115 Bonn, Germany.
Klaus Willecke

Institut fuer Genetik, Rheinische Friedrich-Wilhelms-Universitaet, D-53115 Bonn, Germany.
Takahiro Takano

Center for Translational Neuromedicine, Division of Glial Disease and Therapeutics, University of Rochester Medical School, 601 Elmwood Avenue, Rochester, NY 14642, USA.
Maiken Nedergaard

Center for Translational Neuromedicine, Division of Glial Disease and Therapeutics, University of Rochester Medical School, 601 Elmwood Avenue, Rochester, NY 14642, USA.

Description

<jats:p>Local decreases in extracellular calcium trigger signaling events in astrocytes that activate inhibitory neurotransmission.</jats:p>

Science Signaling

Science Signaling 5 (208), ra8-, 2012-01-24

American Association for the Advancement of Science (AAAS)