Extracellular Ca <sup>2+</sup> Acts as a Mediator of Communication from Neurons to Glia
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- Arnulfo Torres
- Center for Translational Neuromedicine, Division of Glial Disease and Therapeutics, University of Rochester Medical School, 601 Elmwood Avenue, Rochester, NY 14642, USA.
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- Fushun Wang
- Center for Translational Neuromedicine, Division of Glial Disease and Therapeutics, University of Rochester Medical School, 601 Elmwood Avenue, Rochester, NY 14642, USA.
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- Qiwu Xu
- Center for Translational Neuromedicine, Division of Glial Disease and Therapeutics, University of Rochester Medical School, 601 Elmwood Avenue, Rochester, NY 14642, USA.
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- Takumi Fujita
- Center for Translational Neuromedicine, Division of Glial Disease and Therapeutics, University of Rochester Medical School, 601 Elmwood Avenue, Rochester, NY 14642, USA.
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- Radoslaw Dobrowolski
- Institut fuer Genetik, Rheinische Friedrich-Wilhelms-Universitaet, D-53115 Bonn, Germany.
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- Klaus Willecke
- Institut fuer Genetik, Rheinische Friedrich-Wilhelms-Universitaet, D-53115 Bonn, Germany.
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- Takahiro Takano
- Center for Translational Neuromedicine, Division of Glial Disease and Therapeutics, University of Rochester Medical School, 601 Elmwood Avenue, Rochester, NY 14642, USA.
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- Maiken Nedergaard
- Center for Translational Neuromedicine, Division of Glial Disease and Therapeutics, University of Rochester Medical School, 601 Elmwood Avenue, Rochester, NY 14642, USA.
説明
<jats:p>Local decreases in extracellular calcium trigger signaling events in astrocytes that activate inhibitory neurotransmission.</jats:p>
収録刊行物
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- Science Signaling
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Science Signaling 5 (208), ra8-, 2012-01-24
American Association for the Advancement of Science (AAAS)