Long-term Severe Impairment of Speech Sound Production in a Patient with Aphemia.

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Other Title
  • 重篤な語音産生障害が長期に及んだ純粋語唖症例

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Description

    We gave speech therapy for a period of 17 months to an 80 year old aphemic patient. According to the computed tomography, the site of the lesion of the cerebral infarction was limited to the perirolandic area. At the onset, the only speech sound the patient could produce voluntarily was [ə]. He could not imitate any speech sounds or any movements of the speech organs asked by the therapist. He also had bucco-facial apraxia. However, his listening and comprehension, as well as his writing and reading remained close to intact. He did not produce any automatic speech and in comparing the difficulty of speech sound production, we found no difference between his automatic-reflective speech and his intentional-purposeful speech. His speech therapy program consisted mainly of articulation treatment and writing. Despite the fact that he had recovered from his bucco-facial apraxia, the articulation treatment was not so effective. 2 hypotheses try to explain the compensatory mechanism for articulatory disturbances : Nielsen suggested that the homologous area in the minor hemisphere does the compensation and Levine et al. explain that the source of the compensation is the residual region around the Broca's area in the dominant hemisphere. However the clinical findings of the present case do not support either of these 2 hypotheses.

Journal

  • Higher Brain Function Research

    Higher Brain Function Research 17 (4), 313-318, 1997

    Japanese Society of Aphasiology (Renamed as Japan Society for Higher Brain Function)

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